Nicole M. Ashpole, Ph.D.
Affiliations: | 2012 | Neuroscience | Indiana University, Bloomington, Bloomington, IN, United States |
Area:
Neuroscience Biology, Genetics, General BiologyGoogle:
"Nicole Ashpole"Mean distance: 53433
Parents
Sign in to add mentor| Andy Hudmon | grad student | 2012 | Indiana University | |
| (The effects of CaMKII signaling on neuronal viability.) | ||||
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Publications
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| Prabhu D, Khan S, Blackburn K, et al. (2019) Loss of Insulin-like Growth Factor-1 Signaling in Astrocytes Disrupts Glutamate Handling. Journal of Neurochemistry |
| Tarantini S, Valcarcel-Ares NM, Yabluchanskiy A, et al. (2017) Insulin-like growth factor 1 deficiency exacerbates hypertension-induced cerebral microhemorrhages in mice, mimicking the aging phenotype. Aging Cell |
| Zhang D, Marlin MC, Liang Z, et al. (2016) Protein Tyrosine Phosphatase MEG2 Regulates the Transport and Signal Transduction of Tropomyosin Receptor Kinase A. The Journal of Biological Chemistry |
| Tarantini S, Tucsek Z, Valcarcel-Ares MN, et al. (2016) Circulating IGF-1 deficiency exacerbates hypertension-induced microvascular rarefaction in the mouse hippocampus and retrosplenial cortex: implications for cerebromicrovascular and brain aging. Age (Dordrecht, Netherlands) |
| Toth P, Tarantini S, Ashpole NM, et al. (2015) IGF-1 deficiency impairs neurovascular coupling in mice: implications for cerebromicrovascular aging. Aging Cell |
| Lee WH, Xu Z, Ashpole NM, et al. (2015) Small molecule inhibitors of PSD95-nNOS protein-protein interactions as novel analgesics. Neuropharmacology |
| Toth P, Tarantini S, Tucsek Z, et al. (2014) Resveratrol treatment rescues neurovascular coupling in aged mice: role of improved cerebromicrovascular endothelial function and downregulation of NADPH oxidase. American Journal of Physiology. Heart and Circulatory Physiology. 306: H299-308 |
| Ashpole NM, Chawla AR, Martin MP, et al. (2013) Loss of calcium/calmodulin-dependent protein kinase II activity in cortical astrocytes decreases glutamate uptake and induces neurotoxic release of ATP. The Journal of Biological Chemistry. 288: 14599-611 |
| Song W, Xiao Y, Chen H, et al. (2012) The human Nav1.5 F1486 deletion associated with long QT syndrome leads to impaired sodium channel inactivation and reduced lidocaine sensitivity. The Journal of Physiology. 590: 5123-39 |
| Ashpole NM, Herren AW, Ginsburg KS, et al. (2012) Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1.5 gating by multiple phosphorylation sites. The Journal of Biological Chemistry. 287: 19856-69 |