Calvin S. Carter, Ph.D.
Affiliations: | 2014 | Neuroscience | University of Iowa, Iowa City, IA |
Area:
Neuroscience Biology, Medicine and Surgery, Human DevelopmentGoogle:
"Calvin Carter"Mean distance: 23748
Parents
Sign in to add mentor| Val C. Sheffield | grad student | 2014 | University of Iowa | |
| (Characterizing the role of primary cilia in neural progenitor cell development and neonatal hydrocephalus.) | ||||
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Publications
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| Carter CS, Huang SC, Searby CC, et al. (2022) Exposure to Static Magnetic and Electric Fields Treats Type 2 Diabetes. Cell Metabolism. 34: 1893 |
| Stubbs T, Koemeter-Cox A, Bingman JI, et al. (2022) Disruption of dopamine receptor 1 localization to primary cilia impairs signaling in striatal neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Huang SC, Pak TK, Graber CP, et al. (2022) An open source and convenient method for the wide-spread testing of COVID-19 using deep throat sputum samples. Peerj. 10: e13277 |
| Pak TK, Carter CS, Zhang Q, et al. (2021) A mouse model of Bardet-Biedl Syndrome has impaired fear memory, which is rescued by lithium treatment. Plos Genetics. 17: e1009484 |
| Carter CS, Huang S, Searby C, et al. (2021) Response to: An alternative hypothesis for why exposure to static magnetic and electric fields treats type 2 diabetes. American Journal of Physiology. Endocrinology and Metabolism |
| Carter CS, Huang S, Searby C, et al. (2021) Counterpoint regarding an alternative hypothesis for why exposure to static magnetic and electric fields treat type 2 diabetes. American Journal of Physiology. Endocrinology and Metabolism |
| Carter CS, Huang SC, Searby CC, et al. (2020) Exposure to Static Magnetic and Electric Fields Treats Type 2 Diabetes. Cell Metabolism. 32: 1076 |
| Carter CS, Huang SC, Searby CC, et al. (2020) Exposure to Static Magnetic and Electric Fields Treats Type 2 Diabetes. Cell Metabolism. 32: 561-574.e7 |
| Weihbrecht K, Goar WA, Carter CS, et al. (2018) Genotypic and phenotypic characterization of the Sdccag8Tn(sb-Tyr)2161B.CA1C2Ove mouse model. Plos One. 13: e0192755 |
| Zhang Q, Nishimura D, Vogel T, et al. (2013) BBS7 is required for BBSome formation and its absence in mice results in Bardet-Biedl syndrome phenotypes and selective abnormalities in membrane protein trafficking. Journal of Cell Science. 126: 2372-80 |