Alla Grishok
Affiliations: | Biochemistry and Molecular Biophysics | Columbia University, New York, NY |
Area:
Biochemistry, General Biophysics, GeneticsGoogle:
"Alla Grishok"Mean distance: (not calculated yet)
Children
Sign in to add traineeDaphne C. Avgousti | grad student | 2012 | Columbia |
Lisa M. Kennedy | grad student | 2013 | Columbia |
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Publications
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Sepulveda GP, Gushchanskaia ES, Mora-Martin A, et al. (2024) DOT1L stimulates MYC/Mondo transcription factor activity by promoting its degradation cycle on chromatin. Biorxiv : the Preprint Server For Biology |
Liontis T, Verma K, Grishok A. (2023) DOT-1.1 (DOT1L) deficiency in leads to small RNA-dependent gene activation. Bba Advances. 3: 100080 |
Farley SJ, Grishok A, Zeldich E. (2022) Shaking up the silence: consequences of HMGN1 antagonizing PRC2 in the Down syndrome brain. Epigenetics & Chromatin. 15: 39 |
Ceballos A, Esse R, Grishok A. (2021) The proline-rich domain of MML-1 is biologically important but not required for localization to target promoters. Micropublication Biology. 2021 |
Grishok A. (2021) Small RNAs Worm Up Transgenerational Epigenetics Research. Dna. 1: 37-48 |
Lascarez-Lagunas LI, Herruzo E, Grishok A, et al. (2020) DOT-1.1-dependent H3K79 methylation promotes normal meiotic progression and meiotic checkpoint function in C. elegans. Plos Genetics. 16: e1009171 |
Esse R, Grishok A. (2020) Deficient in DOT-1.1 Exhibit Increases in H3K9me2 at Enhancer and Certain RNAi-Regulated Regions. Cells. 9 |
Zaarur N, Desevin K, Mackenzie J, et al. (2019) ATGL-1 mediates the effect of dietary restriction and the insulin/IGF-1 signaling pathway on longevity in C. elegans. Molecular Metabolism |
Esse R, Gushchanskaia ES, Lord A, et al. (2019) DOT1L complex suppresses transcription from enhancer elements and ectopic RNAi in . Rna (New York, N.Y.) |
Gushchanskaia ES, Esse R, Ma Q, et al. (2019) Interplay between small RNA pathways shapes chromatin landscapes in C. elegans. Nucleic Acids Research. 47: 5603-5616 |