Oliver Wilhelm Weinmann
Affiliations: | Brain Research Institute Zurich, Zürich, Zürich, Switzerland |
Area:
Neuroscience, Electron Microscopy, ImmunohistochemistryGoogle:
"Oliver Weinmann"Mean distance: 15.2 (cluster 6) | S | N | B | C | P |
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Publications
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Rust R, Holm MM, Egger M, et al. (2023) Nogo-A is secreted in extracellular vesicles, occurs in blood and can influence vascular permeability. Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism. 271678X231216270 |
Correa D, Scheuber MI, Shan H, et al. (2023) Intranasal delivery of full-length anti-Nogo-A antibody: A potential alternative route for therapeutic antibodies to central nervous system targets. Proceedings of the National Academy of Sciences of the United States of America. 120: e2200057120 |
Stehle JH, Sheng Z, Hausmann L, et al. (2021) Exercise-induced Nogo-A influences rodent motor learning in a time-dependent manner. Plos One. 16: e0250743 |
Schneider MP, Sartori AM, Ineichen BV, et al. (2019) Anti-Nogo-A antibodies as a potential causal therapy for lower urinary tract dysfunction after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Seyedsadr MS, Weinmann O, Amorim A, et al. (2018) Inactivation of sphingosine-1-phosphate receptor 2 (S1PR2) decreases demyelination and enhances remyelination in animal models of multiple sclerosis. Neurobiology of Disease. 124: 189-201 |
Berry S, Weinmann O, Fritz AK, et al. (2018) Loss of Nogo-A, encoded by the schizophrenia risk gene Rtn4, reduces mGlu3 expression and causes hyperexcitability in hippocampal CA3 circuits. Plos One. 13: e0200896 |
Berry S, Weinmann O, Fritz A, et al. (2018) Loss of Nogo-A, encoded by the schizophrenia risk gene Rtn4, reduces mGlu3 expression Plos One. 13 |
Zemmar A, Chen CC, Weinmann O, et al. (2017) Oligodendrocyte- and Neuron-Specific Nogo-A Restrict Dendritic Branching and Spine Density in the Adult Mouse Motor Cortex. Cerebral Cortex (New York, N.Y. : 1991). 1-9 |
Ineichen BV, Weinmann O, Good N, et al. (2016) Sudan black: a fast, easy, and non-toxic method to assess myelin repair in demyelinating diseases. Neuropathology and Applied Neurobiology |
Filli L, Engmann AK, Zörner B, et al. (2014) Bridging the gap: a reticulo-propriospinal detour bypassing an incomplete spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 13399-410 |