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Roger M. Nitsch, MD

Psychiatry Universität Zürich, Zürich, ZH, Switzerland 
Alzheimer’s disease, genetics
"Roger Nitsch"

Mean distance: 14.64 (cluster 11)
Cross-listing: Alzheimer's Tree

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de Leeuw SM, Kirschner AWT, Lindner K, et al. (2021) APOE2, E3, and E4 differentially modulate cellular homeostasis, cholesterol metabolism, and inflammatory response in isogenic iPSC-derived astrocytes. Stem Cell Reports
Michalon A, Hagenbuch A, Huy C, et al. (2021) A human antibody selective for transthyretin amyloid removes cardiac amyloid through phagocytic immune cells. Nature Communications. 12: 3142
Massalimova A, Ni R, Nitsch RM, et al. (2021) Diffusion Tensor Imaging Reveals Whole-Brain Microstructural Changes in the P301L Mouse Model of Tauopathy. Neuro-Degenerative Diseases. 1-12
Probst S, Riese F, Kägi L, et al. (2021) Lysine acetyltransferase Tip60 acetylates the APP adaptor Fe65 to increase its transcriptional activity. Biological Chemistry. 402: 481-499
Treyer V, Meyer RS, Buchmann A, et al. (2021) Physical activity is associated with lower cerebral beta-amyloid and cognitive function benefits from lifetime experience-a study in exceptional aging. Plos One. 16: e0247225
Meier IB, Lao PJ, Gietl A, et al. (2020) Brain areas with normatively greater cerebral perfusion in early life may be more susceptible to beta amyloid deposition in late life. Cerebral Circulation - Cognition and Behavior. 1
Tackenberg C, Kulic L, Nitsch RM. (2020) Familial Alzheimer's disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system. Plos One. 15: e0239584
Kirabali T, Rust R, Rigotti S, et al. (2020) Distinct changes in all major components of the neurovascular unit across different neuropathological stages of Alzheimer's disease. Brain Pathology (Zurich, Switzerland)
Probst S, Krüger M, Kägi L, et al. (2020) Fe65 is the sole family member mediating transcription regulated by the amyloid precursor protein. Journal of Cell Science
Gericke C, Mallone A, Engelhardt B, et al. (2020) Oligomeric Forms of Human Amyloid-Beta(1-42) Inhibit Antigen Presentation. Frontiers in Immunology. 11: 1029
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