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Roger M. Nitsch, MD

Affiliations: 
Psychiatry Universität Zürich, Zürich, ZH, Switzerland 
Area:
Alzheimer’s disease, genetics
Website:
http://www.dpr.uzh.ch/aboutus/People/directors/nitsch.html
Google:
"Roger Nitsch"
Bio:

http://www.researchprofiles.collexis.com/jad/expert.asp?u_id=96

http://www.neurimmune.com/index.php?page=187

Mean distance: 14.64 (cluster 11)
 
SNBCP
Cross-listing: Alzheimer's Tree

BETA: Related publications

Publications

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Rust R, Weber RZ, Generali M, et al. (2022) Xeno-free induced pluripotent stem cell-derived neural progenitor cells for in vivo applications. Journal of Translational Medicine. 20: 421
de Leeuw SM, Kirschner AWT, Lindner K, et al. (2022) APOE2, E3, and E4 differentially modulate cellular homeostasis, cholesterol metabolism, and inflammatory response in isogenic iPSC-derived astrocytes. Stem Cell Reports. 17: 1229-1231
Weber RZ, Bodenmann C, Uhr D, et al. (2022) Intracerebral Transplantation and In Vivo Bioluminescence Tracking of Human Neural Progenitor Cells in the Mouse Brain. Journal of Visualized Experiments : Jove
de Leeuw SM, Kirschner AWT, Lindner K, et al. (2021) APOE2, E3, and E4 differentially modulate cellular homeostasis, cholesterol metabolism, and inflammatory response in isogenic iPSC-derived astrocytes. Stem Cell Reports
Tackenberg C, Kulic L, Nitsch RM. (2020) Familial Alzheimer's disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system. Plos One. 15: e0239584
Kirabali T, Rust R, Rigotti S, et al. (2020) Distinct changes in all major components of the neurovascular unit across different neuropathological stages of Alzheimer's disease. Brain Pathology (Zurich, Switzerland)
Gericke C, Mallone A, Engelhardt B, et al. (2020) Oligomeric Forms of Human Amyloid-Beta(1-42) Inhibit Antigen Presentation. Frontiers in Immunology. 11: 1029
Rust R, Kirabali T, Grönnert L, et al. (2020) A Practical Guide to the Automated Analysis of Vascular Growth, Maturation and Injury in the Brain. Frontiers in Neuroscience. 14: 244
Kirabali T, Rigotti S, Siccoli A, et al. (2019) The amyloid-β degradation intermediate Aβ34 is pericyte-associated and reduced in brain capillaries of patients with Alzheimer's disease. Acta Neuropathologica Communications. 7: 194
Tackenberg C, Nitsch RM. (2019) The secreted APP ectodomain sAPPα, but not sAPPβ, protects neurons against Aβ oligomer-induced dendritic spine loss and increased tau phosphorylation. Molecular Brain. 12: 27
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