Indrapal N. Singh
Affiliations: | 2002-2006 | Anatomy & Neurobiology | University of Kentucky College of Medicine, Lexington, KY, United States |
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"Indrapal Singh"Mean distance: 19.8 (cluster 32)
Parents
Sign in to add mentor| Kurt F. Hauser | research scientist | 2002-2006 | University of Kentucky College of Medicine |
Collaborators
Sign in to add collaborator| Edward D. Hall | collaborator | 2004- | University of Kentucky (Neurotrauma Tree) |
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Publications
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| Hill RL, Singh IN, Wang JA, et al. (2020) Protective effects of phenelzine administration on synaptic and non-synaptic cortical mitochondrial function and lipid peroxidation-mediated oxidative damage following TBI in young adult male rats. Experimental Neurology. 113322 |
| Hill RL, Singh IN, Brelsfoard J, et al. (2020) Pharmacological inhibition of lipid peroxidative damage by the 21-aminosteroid U-74389G improves cortical mitochondrial function following traumatic brain injury in young adult male rats. Neuropharmacology. 108023 |
| Hill RL, Singh IN, Wang J, et al. (2018) THE EFFECTS OF PHENELZINE ADMINISTRATION OF MITOCHONDRIAL FUNCTION, CALCIUM HANDLING AND CYTOSKELETAL DEGRADATION FOLLOWING EXPERIMENTAL TRAUMATIC BRAIN INJURY. Journal of Neurotrauma |
| Hill RL, Kulbe JR, Singh IN, et al. (2018) Synaptic mitochondria are more susceptible to traumatic brain injury-induced oxidative damage and respiratory dysfunction than non-synaptic mitochondria. Neuroscience |
| Kulbe JR, Singh IN, Wang J, et al. (2018) CONTINUOUS INFUSION OF PHENELZINE, CYCLOSPORINE A OR THE COMBINATION: EVALUATION OF MITOCHONDRIAL BIOENERGETICS, OXIDATIVE DAMAGE AND CYTOSKELETAL DEGRADATION FOLLOWING SEVERE CONTROLLED CORTICAL IMPACT TRAUMATIC BRAIN INJURY IN RATS. Journal of Neurotrauma |
| Hill RL, Singh IN, Wang JA, et al. (2017) Time courses of post-injury mitochondrial oxidative damage and respiratory dysfunction and neuronal cytoskeletal degradation in a rat model of focal traumatic brain injury. Neurochemistry International |
| Cebak JE, Singh IN, Hill RL, et al. (2016) PHENELZINE PROTECTS BRAIN MITOCHONDRIAL FUNCTION In vitro and In vivo FOLLOWING TRAUMATIC BRAIN INJURY BY SCAVENGING THE REACTIVE CARBONYLS 4-HYDROXYNONENAL AND ACROLEIN LEADING TO CORTICAL HISTOLOGICAL NEUROPROTECTION. Journal of Neurotrauma |
| Kulbe JR, Hill RL, Singh IN, et al. (2016) SYNAPTIC MITOCHONDRIA SUSTAIN MORE DAMAGE THAN NON-SYNAPTIC MITOCHONDRIA FOLLOWING TRAUMATIC BRAIN INJURY AND ARE PROTECTED BY CYCLOSPORINE A. Journal of Neurotrauma |
| Hall ED, Wang JA, Bosken JM, et al. (2015) Lipid peroxidation in brain or spinal cord mitochondria after injury. Journal of Bioenergetics and Biomembranes |
| Miller DM, Singh IN, Wang JA, et al. (2015) Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice. Experimental Neurology. 264: 103-10 |