R. John Mayer, PhD
Affiliations: | School of Biomedical Sciences | University of Nottingham, Nottingham, England, United Kingdom |
Website:
http://www.google.com/imgres?imgurl=http://www.nottingham.ac.uk/neuroscience/images/staff/j_mayer.jpg&imgrefurl=http://www.nottingham.ac.uk/neuroscience/contact/a-z/M-R/mayer_john.phtml&usg=__3loRcZYgMeFnT9X_elalyg4DUOQ=&h=314&w=239&sz=68&hl=en&start=35&zGoogle:
"R. Mayer"Bio:
http://www.nature.com/nrm/journal/v6/n8/abs/nrm1700.html
Mean distance: 106866
Cross-listing: Neuropathology Tree
Children
Sign in to add traineeDavid G Fernig | grad student | 1981-1985 | Nottingham |
David G Fernig | post-doc | 1985-1988 | Nottingham |
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Publications
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Kageyama S, Sou YS, Uemura T, et al. (2014) Proteasome dysfunction activates autophagy and the Keap1-Nrf2 pathway. The Journal of Biological Chemistry. 289: 24944-55 |
Elkharaz J, Ugun-Klusek A, Constantin-Teodosiu D, et al. (2013) Implications for oxidative stress and astrocytes following 26S proteasomal depletion in mouse forebrain neurones. Biochimica Et Biophysica Acta. 1832: 1930-8 |
Paine SM, Anderson G, Bedford K, et al. (2013) Pale body-like inclusion formation and neurodegeneration following depletion of 26S proteasomes in mouse brain neurones are independent of α-synuclein. Plos One. 8: e54711 |
Rezvani N, Elkharaz J, Lawler K, et al. (2012) Heterozygosity for the proteasomal Psmc1 ATPase is insufficient to cause neuropathology in mouse brain, but causes cell cycle defects in mouse embryonic fibroblasts. Neuroscience Letters. 521: 130-5 |
Paine S, Lowe J, Bedford L, et al. (2011) Can neurodegeneration be separated from neuropathological hallmarks of chronic idiopathic human neurodegenerative disease? A perspective from modelling! Biochemical Society Transactions. 39: 917-9 |
Bedford L, Layfield R, Mayer RJ, et al. (2011) Diverse polyubiquitin chains accumulate following 26S proteasomal dysfunction in mammalian neurones. Neuroscience Letters. 491: 44-7 |
Bedford L, Lowe J, Dick LR, et al. (2011) Ubiquitin-like protein conjugation and the ubiquitin-proteasome system as drug targets. Nature Reviews. Drug Discovery. 10: 29-46 |
Bedford L, Paine S, Sheppard PW, et al. (2010) Assembly, structure, and function of the 26S proteasome. Trends in Cell Biology. 20: 391-401 |
Paine S, Bedford L, Thorpe JR, et al. (2009) Immunoreactivity to Lys63-linked polyubiquitin is a feature of neurodegeneration. Neuroscience Letters. 460: 205-8 |
Bedford L, Paine S, Rezvani N, et al. (2009) The UPS and autophagy in chronic neurodegenerative disease: six of one and half a dozen of the other--or not? Autophagy. 5: 224-7 |