Jeff W. Pollard, Ph.D.

Affiliations: 
Developmental and Molecular Biology Albert Einstein College of Medicine, New York, New York, United States 
Area:
tumor progression and metastasis
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"Jeff Pollard"
Mean distance: 18.57 (cluster 18)
 
SNBCP
Cross-listing: Chemistry Tree

Parents

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Cliff Stanners post-doc 1975-1980 McGill

Children

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Paula E. Cohen post-doc 1993-1998 Edinburgh (Cell Biology Tree)
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Publications

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Cohen PE, Pollack SE, Pollard JW. (2006) Genetic analysis of chromosome pairing, recombination, and cell cycle control during first meiotic prophase in mammals. Endocrine Reviews. 27: 398-426
Cohen PE, Zhu L, Nishimura K, et al. (2002) Colony-stimulating factor 1 regulation of neuroendocrine pathways that control gonadal function in mice. Endocrinology. 143: 1413-22
Cohen PE, Pollard JW. (2001) Regulation of meiotic recombination and prophase I progression in mammals. Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology. 23: 996-1009
Zindy F, den Besten W, Chen B, et al. (2001) Control of spermatogenesis in mice by the cyclin D-dependent kinase inhibitors p18(Ink4c) and p19(Ink4d). Molecular and Cellular Biology. 21: 3244-55
Kneitz B, Cohen PE, Avdievich E, et al. (2000) MutS homolog 4 localization to meiotic chromosomes is required for chromosome pairing during meiosis in male and female mice. Genes & Development. 14: 1085-97
Gouon-Evans V, Rothenberg ME, Pollard JW. (2000) Postnatal mammary gland development requires macrophages and eosinophils. Development (Cambridge, England). 127: 2269-82
Cohen PE, Nishimura K, Zhu L, et al. (1999) Macrophages: important accessory cells for reproductive function. Journal of Leukocyte Biology. 66: 765-72
Edelmann W, Cohen PE, Kneitz B, et al. (1999) Mammalian MutS homologue 5 is required for chromosome pairing in meiosis. Nature Genetics. 21: 123-7
Cohen PE, Pollard JW. (1998) Normal sexual function in male mice lacking a functional type I interleukin-1 (IL-1) receptor. Endocrinology. 139: 815-8
Edelmann W, Yang K, Umar A, et al. (1997) Mutation in the mismatch repair gene Msh6 causes cancer susceptibility. Cell. 91: 467-77
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