Joshua P. Whitt
Affiliations: | Physiology | University of Maryland School of Medicine, Baltimore, MD, United States |
Area:
Ion channels, Alternative SplicingGoogle:
"Joshua Whitt"Mean distance: 15.17 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentor| Andrea Meredith | grad student | 2011-2016 | University of Maryland Medical School |
| Diane Lipscombe | post-doc | 2016- | Brown |
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Publications
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| Dominov JA, Madigan LA, Whitt JP, et al. (2023) Up-regulation of cholesterol synthesis pathways and limited neurodegeneration in a knock-in mutant mouse model of ALS. Biorxiv : the Preprint Server For Biology |
| Plante AE, Whitt JP, Meredith AL. (2021) BK channel activation by L-type Ca channels Ca1.2 and Ca1.3 during the subthreshold phase of an action potential. Journal of Neurophysiology |
| Yanagi KS, Wu Z, Amaya J, et al. (2019) Meta-analysis of Genetic Modifiers Reveals Candidate Dysregulated Pathways in Amyotrophic Lateral Sclerosis. Neuroscience. 396: A3-A20 |
| Whitt J, McNally B, Harvey J, et al. (2018) BK Channels are Activated by Distinct Calcium Sources during Day and Night in the Circadian Clock Biophysical Journal. 114: 132a |
| Whitt J, Harvey J, Meredith A. (2018) β2 is Not Required for Daytime Activation of BK Currents by L-Type Calcium Channels Biophysical Journal. 114: 131a-132a |
| Whitt JP, McNally BA, Meredith AL. (2017) Differential contribution of Ca2+ sources to day and night BK current activation in the circadian clock. The Journal of General Physiology |
| Meredith A, Montgomery JR, Whitt JP. (2017) BK Channels: Sensors that Switch Membranes between Day and Night States in the Circadian Clock Biophysical Journal. 112: 8a |
| Whitt JP, Montgomery JR, Meredith AL. (2016) BK channel inactivation gates daytime excitability in the circadian clock. Nature Communications. 7: 10837 |
| Whitt JP, Meredith AL. (2016) BK-Ca2+ Channel Coupling in the Mouse Circadian Clock Biophysical Journal. 110: 114a |
| Zemen BG, Lai MH, Whitt JP, et al. (2015) Generation of Kcnma1fl-tdTomato, a conditional deletion of the BK channel α subunit in mouse. Physiological Reports. 3 |