K Ulrich Ulli Bayer
Affiliations: | UCHSC, Aurora, CO, United States |
Area:
Synaptic plasticity, Down syndrome, Schizophrenia, Addiction, Stroke, Global cerebral ischemiaWebsite:
http://www.ucdenver.edu/academics/colleges/medicalschool/departments/Pharmacology/faculty/Pages/bayer.aspxGoogle:
"K Bayer"Mean distance: 15.17 (cluster 11) | S | N | B | C | P |
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Publications
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Tullis JE, Larsen ME, Rumian NL, et al. (2023) LTP induction by structural rather than enzymatic functions of CaMKII. Nature |
Rumian NL, Freund RK, Dell'Acqua ML, et al. (2023) Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice. Science Signaling. 16: eade5892 |
Tullis JE, Bayer KU. (2023) Distinct synaptic pools of DAPK1 differentially regulate activity-dependent synaptic CaMKII accumulation. Iscience. 26: 106723 |
Larsen ME, Buonarati OR, Qian H, et al. (2023) Stimulating β-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP mode. The Journal of Biological Chemistry. 104706 |
Cook SG, Rumian NL, Bayer KU. (2022) CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity. The Journal of Biological Chemistry. 102299 |
Brown CN, Rumian NL, Tullis JE, et al. (2022) Aβ-induced synaptic impairments require CaMKII activity that is stimulated by indirect signaling events. Iscience. 25: 104368 |
Tullis JE, Buonarati OR, Coultrap SJ, et al. (2021) GluN2B S1303 phosphorylation by CaMKII or DAPK1: no indication for involvement in ischemia or LTP. Iscience. 24: 103214 |
Brown CN, Cook SG, Allen HF, et al. (2021) Characterization of six CaMKIIα variants found in patients with schizophrenia. Iscience. 24: 103184 |
Goodell DJ, Tullis JE, Bayer KU. (2021) Young DAPK1 knockout mice have altered pre-synaptic function. Journal of Neurophysiology |
Cook SG, Buonarati OR, Coultrap SJ, et al. (2021) CaMKII holoenzyme mechanisms that govern the LTP versus LTD decision. Science Advances. 7 |