K Ulrich Ulli Bayer

Affiliations: 
UCHSC, Aurora, CO, United States 
Area:
Synaptic plasticity, Down syndrome, Schizophrenia, Addiction, Stroke, Global cerebral ischemia
Website:
http://www.ucdenver.edu/academics/colleges/medicalschool/departments/Pharmacology/faculty/Pages/bayer.aspx
Google:
"K Bayer"
Mean distance: 15.17 (cluster 11)
 
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Publications

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Tullis JE, Larsen ME, Rumian NL, et al. (2023) LTP induction by structural rather than enzymatic functions of CaMKII. Nature
Rumian NL, Freund RK, Dell'Acqua ML, et al. (2023) Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice. Science Signaling. 16: eade5892
Tullis JE, Bayer KU. (2023) Distinct synaptic pools of DAPK1 differentially regulate activity-dependent synaptic CaMKII accumulation. Iscience. 26: 106723
Larsen ME, Buonarati OR, Qian H, et al. (2023) Stimulating β-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP mode. The Journal of Biological Chemistry. 104706
Cook SG, Rumian NL, Bayer KU. (2022) CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity. The Journal of Biological Chemistry. 102299
Brown CN, Rumian NL, Tullis JE, et al. (2022) Aβ-induced synaptic impairments require CaMKII activity that is stimulated by indirect signaling events. Iscience. 25: 104368
Tullis JE, Buonarati OR, Coultrap SJ, et al. (2021) GluN2B S1303 phosphorylation by CaMKII or DAPK1: no indication for involvement in ischemia or LTP. Iscience. 24: 103214
Brown CN, Cook SG, Allen HF, et al. (2021) Characterization of six CaMKIIα variants found in patients with schizophrenia. Iscience. 24: 103184
Goodell DJ, Tullis JE, Bayer KU. (2021) Young DAPK1 knockout mice have altered pre-synaptic function. Journal of Neurophysiology
Cook SG, Buonarati OR, Coultrap SJ, et al. (2021) CaMKII holoenzyme mechanisms that govern the LTP versus LTD decision. Science Advances. 7
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