Gary W. Arendash

University of South Florida, Tampa, FL, United States 
Neuroscience Biology
"Gary Arendash"
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Citron BA, Saykally JN, Cao C, et al. (2015) Transcription factor Sp1 inhibition, memory, and cytokines in a mouse model of Alzheimer's disease. American Journal of Neurodegenerative Disease. 4: 40-8
Citron BA, Saykally JN, Cao C, et al. (2015) Transcription factor Sp1 inhibition, memory, and cytokines in a mouse model of alzheimer’s disease American Journal of Neurodegenerative Diseases. 4: 40-48
Tajiri N, Kellogg SL, Shimizu T, et al. (2013) Traumatic brain injury precipitates cognitive impairment and extracellular Aβ aggregation in Alzheimer's disease transgenic mice. Plos One. 8: e78851
Dragicevic N, Delic V, Cao C, et al. (2012) Caffeine increases mitochondrial function and blocks melatonin signaling to mitochondria in Alzheimer's mice and cells. Neuropharmacology. 63: 1368-79
Arendash GW. (2012) Transcranial electromagnetic treatment against Alzheimer's disease: why it has the potential to trump Alzheimer's disease drug development. Journal of Alzheimer's Disease : Jad. 32: 243-66
Arendash GW, Mori T, Dorsey M, et al. (2012) Electromagnetic treatment to old Alzheimer's mice reverses β-amyloid deposition, modifies cerebral blood flow, and provides selected cognitive benefit. Plos One. 7: e35751
Cao C, Loewenstein DA, Lin X, et al. (2012) High Blood caffeine levels in MCI linked to lack of progression to dementia. Journal of Alzheimer's Disease : Jad. 30: 559-72
Mori T, Rezai-Zadeh K, Koyama N, et al. (2012) Tannic acid is a natural β-secretase inhibitor that prevents cognitive impairment and mitigates Alzheimer-like pathology in transgenic mice. The Journal of Biological Chemistry. 287: 6912-27
Dragicevic N, Bradshaw PC, Mamcarz M, et al. (2011) Long-term electromagnetic field treatment enhances brain mitochondrial function of both Alzheimer's transgenic mice and normal mice: A mechanism for electromagnetic field-induced cognitive benefit? Neuroscience. 185: 135-149
Dragicevic N, Copes N, O'Neal-Moffitt G, et al. (2011) Melatonin treatment restores mitochondrial function in Alzheimer's mice: a mitochondrial protective role of melatonin membrane receptor signaling. Journal of Pineal Research. 51: 75-86
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