Fenghua Hu
Affiliations: | Cornell University, Ithaca, NY, United States |
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| Feng T, Mai S, Roscoe JM, et al. (2020) Loss of TMEM106B and PGRN leads to severe lysosomal abnormalities and neurodegeneration in mice. Embo Reports. e50219 |
| Feng T, Sheng RR, Solé-Domènech S, et al. (2020) A role of the frontotemporal lobar degeneration risk factor TMEM106B in myelination. Brain : a Journal of Neurology |
| Zhou X, Paushter DH, Pagan MD, et al. (2019) Progranulin deficiency leads to reduced glucocerebrosidase activity. Plos One. 14: e0212382 |
| Lan Y, Sullivan PM, Hu F. (2019) SMCR8 negatively regulates AKT and MTORC1 signaling to modulate lysosome biogenesis and tissue homeostasis. Autophagy. 1-15 |
| Zhou X, Sullivan PM, Paushter DH, et al. (2018) The Interaction Between Progranulin with Sortilin and the Lysosome. Methods in Molecular Biology (Clifton, N.J.). 1806: 269-288 |
| Nicholson AM, Zhou X, Perkerson RB, et al. (2018) Loss of Tmem106b is unable to ameliorate frontotemporal dementia-like phenotypes in an AAV mouse model of C9ORF72-repeat induced toxicity. Acta Neuropathologica Communications. 6: 42 |
| Paushter DH, Du H, Feng T, et al. (2018) The lysosomal function of progranulin, a guardian against neurodegeneration. Acta Neuropathologica |
| Zhou X, Sullivan PM, Sun L, et al. (2017) The interaction between progranulin and prosaposin is mediated by granulins and the linker region between saposin B and C. Journal of Neurochemistry |
| Zhou X, Sun L, Bracko O, et al. (2017) Impaired prosaposin lysosomal trafficking in frontotemporal lobar degeneration due to progranulin mutations. Nature Communications. 8: 15277 |
| Zhou X, Sun L, Brady OA, et al. (2017) Elevated TMEM106B levels exaggerate lipofuscin accumulation and lysosomal dysfunction in aged mice with progranulin deficiency. Acta Neuropathologica Communications. 5: 9 |