Carol Colton

Affiliations: 
Georgetown University Medical Center, Washington, DC, United States 
Area:
Neuroscience Biology
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"Carol Colton"
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Publications

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Brown CM, Bushnell CD, Samsa GP, et al. (2015) Chronic Systemic Immune Dysfunction in African-Americans with Small Vessel-Type Ischemic Stroke. Translational Stroke Research
Kan MJ, Lee JE, Wilson JG, et al. (2015) Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 5969-82
Hoos MD, Vitek MP, Ridnour LA, et al. (2014) The impact of human and mouse differences in NOS2 gene expression on the brain's redox and immune environment. Molecular Neurodegeneration. 9: 50
Colton CA, Wilson JG, Everhart A, et al. (2014) mNos2 deletion and human NOS2 replacement in Alzheimer disease models. Journal of Neuropathology and Experimental Neurology. 73: 752-69
Hoos MD, Richardson BM, Foster MW, et al. (2013) Longitudinal study of differential protein expression in an Alzheimer's mouse model lacking inducible nitric oxide synthase. Journal of Proteome Research. 12: 4462-77
Colton CA. (2013) Microglial-neuronal interactions during neurodegenerative diseases. Journal of Neuroimmune Pharmacology : the Official Journal of the Society On Neuroimmune Pharmacology. 8: 4-6
Colton CA. (2013) Immune heterogeneity in neuroinflammation: dendritic cells in the brain. Journal of Neuroimmune Pharmacology : the Official Journal of the Society On Neuroimmune Pharmacology. 8: 145-62
Ridnour LA, Dhanapal S, Hoos M, et al. (2012) Nitric oxide-mediated regulation of β-amyloid clearance via alterations of MMP-9/TIMP-1. Journal of Neurochemistry. 123: 736-49
Xu F, Vitek MP, Colton CA, et al. (2012) Human apolipoprotein E2 promotes parenchymal amyloid deposition and neuronal loss in vasculotropic mutant amyloid-β protein Tg-SwDI mice. Journal of Alzheimer's Disease : Jad. 31: 359-69
Sudduth TL, Wilson JG, Everhart A, et al. (2012) Lithium treatment of APPSwDI/NOS2-/- mice leads to reduced hyperphosphorylated tau, increased amyloid deposition and altered inflammatory phenotype. Plos One. 7: e31993
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