Charles E. Inturrisi

Weill Cornell Medical College, New York, NY, United States 
Neuroscience Biology, Pharmacology, Molecular Biology
"Charles Inturrisi"
Mean distance: 17811
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Mehta N, Inturrisi CE, Horn SD, et al. (2016) Using Chronic Pain Outcomes Data to Improve Outcomes. Anesthesiology Clinics. 34: 395-408
Inturrisi CE, Gregusa AM. (2015) Mechanisms of opioid tolerance Cancer Pain: From Molecules to Suffering
Xu J, Xu M, Brown T, et al. (2013) Stabilization of the μ-opioid receptor by truncated single transmembrane splice variants through a chaperone-like action. The Journal of Biological Chemistry. 288: 21211-27
Bruehl S, Apkarian AV, Ballantyne JC, et al. (2013) Personalized medicine and opioid analgesic prescribing for chronic pain: opportunities and challenges. The Journal of Pain : Official Journal of the American Pain Society. 14: 103-13
Schierberl K, Hao J, Tropea TF, et al. (2011) Cav1.2 L-type Ca²⁺ channels mediate cocaine-induced GluA1 trafficking in the nucleus accumbens, a long-term adaptation dependent on ventral tegmental area Ca(v)1.3 channels. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 13562-75
Reid MC, Bennett DA, Chen WG, et al. (2011) Improving the pharmacologic management of pain in older adults: identifying the research gaps and methods to address them. Pain Medicine (Malden, Mass.). 12: 1336-57
Foley KM, Fins JJ, Inturrisi CE. (2011) A true believer's flawed analysis. Archives of Internal Medicine. 171: 867-8; author reply
Hunter DA, Barr GA, Amador N, et al. (2011) Estradiol-induced antinociceptive responses on formalin-induced nociception are independent of COX and HPA activation. Synapse (New York, N.Y.). 65: 643-51
Chapman CR, Lipschitz DL, Angst MS, et al. (2010) Opioid pharmacotherapy for chronic non-cancer pain in the United States: a research guideline for developing an evidence-base. The Journal of Pain : Official Journal of the American Pain Society. 11: 807-29
Gregus AM, Inra CN, Giordano TP, et al. (2010) Spinal mediators that may contribute selectively to antinociceptive tolerance but not other effects of morphine as revealed by deletion of GluR5. Neuroscience. 169: 475-87
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