Asako Tosaki
Affiliations: | Laboratory for Structural Neuropathology | RIKEN Brain Science Institute, Wakō-shi, Saitama-ken, Japan |
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Publications
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| Yamanaka T, Miyazaki H, Tosaki A, et al. (2020) Gene expression profiling in neuronal cells identifies a different type of transcriptome modulated by NF-Y. Scientific Reports. 10: 21714 |
| Shimizu H, Tosaki A, Ohsawa N, et al. (2017) Parallel homodimer structures of the extracellular domains of the voltage-gated sodium channel β4 subunit explain its role in cell-cell adhesion. The Journal of Biological Chemistry |
| Yamanaka T, Tosaki A, Miyazaki H, et al. (2016) Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS. Scientific Reports. 6: 34575 |
| Shimizu H, Miyazaki H, Ohsawa N, et al. (2016) Structure-based site-directed photo-crosslinking analyses of multimeric cell-adhesive interactions of voltage-gated sodium channel β subunits. Scientific Reports. 6: 26618 |
| Yamanaka T, Tosaki A, Kurosawa M, et al. (2016) Genome-wide analyses in neuronal cells reveal that USF transcription factors regulate lysosomal gene expression. The Febs Journal |
| Yamanaka T, Wong HK, Tosaki A, et al. (2014) Large-scale RNA interference screening in mammalian cells identifies novel regulators of mutant huntingtin aggregation. Plos One. 9: e93891 |
| Yamanaka T, Tosaki A, Kurosawa M, et al. (2014) NF-Y inactivation causes atypical neurodegeneration characterized by ubiquitin and p62 accumulation and endoplasmic reticulum disorganization. Nature Communications. 5: 3354 |
| Yamanaka T, Tosaki A, Kurosawa M, et al. (2013) Loss of aPKCλ in differentiated neurons disrupts the polarity complex but does not induce obvious neuronal loss or disorientation in mouse brains. Plos One. 8: e84036 |
| Yamanaka T, Tosaki A, Wong HK, et al. (2011) Establishment of method for counting of polyglutamine aggregates by Array Scan reader and its application for shRNA high-throughput screening Neuroscience Research. 71: e192 |
| Yamanaka T, Tosaki A, Miyazaki H, et al. (2010) Mutant huntingtin fragment selectively suppresses Brn-2 POU domain transcription factor to mediate hypothalamic cell dysfunction. Human Molecular Genetics. 19: 2099-112 |