Asako Tosaki

Affiliations: 
Laboratory for Structural Neuropathology RIKEN Brain Science Institute, Wakō-shi, Saitama-ken, Japan 
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"Asako Tosaki"
Mean distance: 106866 (cluster 31)
 
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Publications

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Yamanaka T, Miyazaki H, Tosaki A, et al. (2020) Gene expression profiling in neuronal cells identifies a different type of transcriptome modulated by NF-Y. Scientific Reports. 10: 21714
Shimizu H, Tosaki A, Ohsawa N, et al. (2017) Parallel homodimer structures of the extracellular domains of the voltage-gated sodium channel β4 subunit explain its role in cell-cell adhesion. The Journal of Biological Chemistry
Yamanaka T, Tosaki A, Miyazaki H, et al. (2016) Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS. Scientific Reports. 6: 34575
Shimizu H, Miyazaki H, Ohsawa N, et al. (2016) Structure-based site-directed photo-crosslinking analyses of multimeric cell-adhesive interactions of voltage-gated sodium channel β subunits. Scientific Reports. 6: 26618
Yamanaka T, Tosaki A, Kurosawa M, et al. (2016) Genome-wide analyses in neuronal cells reveal that USF transcription factors regulate lysosomal gene expression. The Febs Journal
Yamanaka T, Wong HK, Tosaki A, et al. (2014) Large-scale RNA interference screening in mammalian cells identifies novel regulators of mutant huntingtin aggregation. Plos One. 9: e93891
Yamanaka T, Tosaki A, Kurosawa M, et al. (2014) NF-Y inactivation causes atypical neurodegeneration characterized by ubiquitin and p62 accumulation and endoplasmic reticulum disorganization. Nature Communications. 5: 3354
Yamanaka T, Tosaki A, Kurosawa M, et al. (2013) Loss of aPKCλ in differentiated neurons disrupts the polarity complex but does not induce obvious neuronal loss or disorientation in mouse brains. Plos One. 8: e84036
Yamanaka T, Tosaki A, Miyazaki H, et al. (2010) Mutant huntingtin fragment selectively suppresses Brn-2 POU domain transcription factor to mediate hypothalamic cell dysfunction. Human Molecular Genetics. 19: 2099-112
Wong HK, Bauer PO, Kurosawa M, et al. (2008) Blocking acid-sensing ion channel 1 alleviates Huntington's disease pathology via an ubiquitin-proteasome system-dependent mechanism. Human Molecular Genetics. 17: 3223-35
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