Chika Washizu
Affiliations: | Laboratory for Structural Neuropathology | RIKEN Brain Science Institute, Wakō-shi, Saitama-ken, Japan |
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| Yanaizu M, Washizu C, Nukina N, et al. (2020) CELF2 regulates the species-specific alternative splicing of TREM2. Scientific Reports. 10: 17995 |
| Kino Y, Washizu C, Kurosawa M, et al. (2016) FUS/TLS acts as an aggregation-dependent modifier of polyglutamine disease model mice. Scientific Reports. 6: 35236 |
| Kino Y, Washizu C, Kurosawa M, et al. (2015) FUS/TLS deficiency causes behavioral and pathological abnormalities distinct from amyotrophic lateral sclerosis. Acta Neuropathologica Communications. 3: 24 |
| Kurosawa M, Matsumoto G, Kino Y, et al. (2015) Depletion of p62 reduces nuclear inclusions and paradoxically ameliorates disease phenotypes in Huntington's model mice. Human Molecular Genetics. 24: 1092-105 |
| Kino Y, Washizu C, Kurosawa M, et al. (2015) Nuclear localization of MBNL1: splicing-mediated autoregulation and repression of repeat-derived aberrant proteins. Human Molecular Genetics. 24: 740-56 |
| Kino Y, Washizu C, Aquilanti E, et al. (2011) Intracellular localization and splicing regulation of FUS/TLS are variably affected by amyotrophic lateral sclerosis-linked mutations. Nucleic Acids Research. 39: 2781-98 |
| Kino Y, Washizu C, Oma Y, et al. (2009) MBNL and CELF proteins regulate alternative splicing of the skeletal muscle chloride channel CLCN1. Nucleic Acids Research. 37: 6477-90 |
| Wong HK, Bauer PO, Kurosawa M, et al. (2008) Blocking acid-sensing ion channel 1 alleviates Huntington's disease pathology via an ubiquitin-proteasome system-dependent mechanism. Human Molecular Genetics. 17: 3223-35 |
| Yamanaka T, Miyazaki H, Oyama F, et al. (2008) Mutant Huntingtin reduces HSP70 expression through the sequestration of NF-Y transcription factor. The Embo Journal. 27: 827-39 |