Roberto Zayas, Ph.D.
Affiliations: | 2005 | University of Minnesota, Twin Cities, Minneapolis, MN |
Area:
Neuroscience Biology, Cell BiologyGoogle:
"Roberto Zayas"Mean distance: 35622
Parents
Sign in to add mentor| Christopher M. Gomez | grad student | 2005 | UMN | |
| (Calcium entry and calcium-mediated mechanisms in the slow -channel syndrome.) | ||||
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| Groshong JS, Spencer MJ, Bhattacharyya BJ, et al. (2007) Calpain activation impairs neuromuscular transmission in a mouse model of the slow-channel myasthenic syndrome. The Journal of Clinical Investigation. 117: 2903-12 |
| Zayas R, Groshong JS, Gomez CM. (2007) Inositol-1,4,5-triphosphate receptors mediate activity-induced synaptic Ca2+ signals in muscle fibers and Ca2+ overload in slow-channel syndrome. Cell Calcium. 41: 343-52 |
| Zayas R, Lasalde-Dominicci J, Gomez CM. (2006) Macroscopic properties of spontaneous mutations in slow-channel syndrome: correlation by domain and disease severity. Synapse (New York, N.Y.). 60: 441-9 |
| Vohra BP, Groshong JS, Zayas R, et al. (2006) Activation of apoptotic pathways at muscle fiber synapses is circumscribed and reversible in a slow-channel syndrome model. Neurobiology of Disease. 23: 462-70 |
| Navedo MF, Lasalde-Dominicci JA, Báez-Pagán CA, et al. (2006) Novel beta subunit mutation causes a slow-channel syndrome by enhancing activation and decreasing the rate of agonist dissociation. Molecular and Cellular Neurosciences. 32: 82-90 |
| Gomez CM, Maselli RA, Groshong J, et al. (2002) Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel syndrome. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 22: 6447-57 |