Kim A. Caldwell

Affiliations: 
The University of Alabama, Tuscaloosa, AL 
Area:
Molecular Biology, Neuroscience Biology
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"Kim Caldwell"
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Publications

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Hartman JH, Gonzalez-Hunt C, Hall SM, et al. (2019) Genetic Defects in Mitochondrial Dynamics in Impact Ultraviolet C Radiation- and 6-hydroxydopamine-Induced Neurodegeneration. International Journal of Molecular Sciences. 20
Gaeta AL, Caldwell KA, Caldwell GA. (2019) Found in Translation: The Utility of Alpha-Synuclein Models of Parkinson's Disease. Brain Sciences. 9
Daniels MJ, Nourse JB, Kim H, et al. (2019) Cyclized NDGA modifies dynamic α-synuclein monomers preventing aggregation and toxicity. Scientific Reports. 9: 2937
Griffin EF, Caldwell KA, Caldwell GA. (2019) Vacuolar protein sorting protein 41 (VPS41) at an intersection of endosomal traffic in neurodegenerative disease. Neural Regeneration Research. 14: 1210-1212
Griffin EF, Scopel SE, Stephen CA, et al. (2019) ApoE-associated modulation of neuroprotection from Aβ-mediated neurodegeneration in transgenic . Disease Models & Mechanisms
Griffin EF, Yan X, Caldwell KA, et al. (2018) Distinct functional roles of Vps41-mediated neuroprotection in Alzheimer's and Parkinson's disease models of neurodegeneration. Human Molecular Genetics. 27: 4176-4193
Caldwell KA, Thies JL, Caldwell GA. (2018) No Country for Old Worms: A Systematic Review of the Application of to Investigate a Bacterial Source of Environmental Neurotoxicity in Parkinson's Disease. Metabolites. 8
Kim H, Perentis RJ, Caldwell GA, et al. (2018) Gene-by-environment interactions that disrupt mitochondrial homeostasis cause neurodegeneration in C. elegans Parkinson's models. Cell Death & Disease. 9: 555
Kim H, Calatayud C, Guha S, et al. (2018) Correction to: The Small GTPase RAC1/CED-10 Is Essential in Maintaining Dopaminergic Neuron Function and Survival Against α-Synuclein-Induced Toxicity. Molecular Neurobiology
Patel D, Xu C, Nagarajan S, et al. (2018) Alpha-synuclein inhibits Snx3-retromer-mediated retrograde recycling of iron transporters in S. cerevisiae and C. elegans models of Parkinson's disease. Human Molecular Genetics
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