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Mihai Covasa

Affiliations: 
Pennsylvania State University, State College, PA, United States 
Area:
Nutrition, Neuroscience Biology
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"Mihai Covasa"
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David M. Savastano grad student 2005 Penn State

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Hamamah S, Hajnal A, Covasa M. (2023) Reduced Striatal Dopamine Transporter Availability and Heightened Response to Natural and Pharmacological Stimulation in CCK-1R-Deficient Obese Rats. International Journal of Molecular Sciences. 24
Duca FA, Swartz TD, Covasa M. (2014) Effect of diet on preference and intake of sucrose in obese prone and resistant rats. Plos One. 9: e111232
Duca FA, Zhong L, Covasa M. (2013) Reduced CCK signaling in obese-prone rats fed a high fat diet. Hormones and Behavior. 64: 812-7
Duca FA, Sakar Y, Covasa M. (2013) The modulatory role of high fat feeding on gastrointestinal signals in obesity. The Journal of Nutritional Biochemistry. 24: 1663-77
Duca FA, Sakar Y, Covasa M. (2013) Combination of obesity and high-fat feeding diminishes sensitivity to GLP-1R agonist exendin-4. Diabetes. 62: 2410-5
Duca FA, Swartz TD, Sakar Y, et al. (2012) Decreased intestinal nutrient response in diet-induced obese rats: role of gut peptides and nutrient receptors. International Journal of Obesity (2005). 37: 375-81
Swartz TD, Duca FA, de Wouters T, et al. (2011) Up-regulation of intestinal type 1 taste receptor 3 and sodium glucose luminal transporter-1 expression and increased sucrose intake in mice lacking gut microbiota. The British Journal of Nutrition. 107: 621-30
Wright J, Campos C, Herzog T, et al. (2011) Reduction of food intake by cholecystokinin requires activation of hindbrain NMDA-type glutamate receptors. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 301: R448-55
Duca F, Swartz T, Covasa M. (2011) Changes in satiation signaling in diet-induced obese (DIO) rats. Diet versus phenotype Appetite. 57: S14
Covasa M. (2010) Deficits in gastrointestinal responses controlling food intake and body weight. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 299: R1423-39
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