Haibing Jiang, Ph.D.
Affiliations: | 2003 | University of Pittsburgh, Pittsburgh, PA, United States |
Area:
PharmacologyGoogle:
"Haibing Jiang"Mean distance: 21373.2
Parents
Sign in to add mentorDonald B. DeFranco | grad student | 2003 | University of Pittsburgh | |
(Mechanisms of polyglutamine expanded huntingtin induced toxicity.) |
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Publications
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Liu J, Li T, Thomas JM, et al. (2016) Synphilin-1 attenuates mutant LRRK2-induced neurodegeneration in Parkinson's disease models. Human Molecular Genetics |
Scharadin TM, Jiang H, Jans R, et al. (2011) TIG3 tumor suppressor-dependent organelle redistribution and apoptosis in skin cancer cells. Plos One. 6: e23230 |
Liang Y, Jiang H, Ratovitski T, et al. (2009) ATF3 plays a protective role against toxicity by N-terminal fragment of mutant huntingtin in stable PC12 cell line. Brain Research. 1286: 221-9 |
Ratovitski T, Gucek M, Jiang H, et al. (2009) Mutant huntingtin N-terminal fragments of specific size mediate aggregation and toxicity in neuronal cells. The Journal of Biological Chemistry. 284: 10855-67 |
Ko HS, Bailey R, Smith WW, et al. (2009) CHIP regulates leucine-rich repeat kinase-2 ubiquitination, degradation, and toxicity. Proceedings of the National Academy of Sciences of the United States of America. 106: 2897-902 |
Eckert RL, Sturniolo MT, Jans R, et al. (2009) TIG3: a regulator of type I transglutaminase activity in epidermis. Amino Acids. 36: 739-46 |
Liu Z, Wang X, Yu Y, et al. (2008) A Drosophila model for LRRK2-linked parkinsonism. Proceedings of the National Academy of Sciences of the United States of America. 105: 2693-8 |
Smith WW, Pei Z, Jiang H, et al. (2006) Kinase activity of mutant LRRK2 mediates neuronal toxicity. Nature Neuroscience. 9: 1231-3 |
Jiang H, Poirier MA, Liang Y, et al. (2006) Depletion of CBP is directly linked with cellular toxicity caused by mutant huntingtin. Neurobiology of Disease. 23: 543-51 |
Smith WW, Pei Z, Jiang H, et al. (2005) Leucine-rich repeat kinase 2 (LRRK2) interacts with parkin, and mutant LRRK2 induces neuronal degeneration. Proceedings of the National Academy of Sciences of the United States of America. 102: 18676-81 |