Joanna L. Jankowsky

Neuroscience Baylor College of Medicine, Houston, TX 
Alzheimer's disease, learning and memory
"Joanna Jankowsky"
Mean distance: 13.39 (cluster 32)
Cross-listing: Alzheimer's Tree - BCM Tree


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Paul H. Patterson grad student 1994-1999 Caltech
David R. Borchelt post-doc 2000-2003 Johns Hopkins Medical School
Henry A. Lester research scientist 2003-2008 Caltech
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Lillehaug S, Yetman MJ, Puchades MA, et al. (2019) Brain-wide distribution of reporter expression in five transgenic tetracycline-transactivator mouse lines. Scientific Data. 6: 190028
Zhao R, Grunke SD, Keralapurath MM, et al. (2016) Impaired Recall of Positional Memory following Chemogenetic Disruption of Place Field Stability. Cell Reports
Yetman MJ, Fowler SW, Jankowsky JL. (2016) Humanized Tau Mice with Regionalized Amyloid Exhibit Behavioral Deficits but No Pathological Interaction. Plos One. 11: e0153724
Lian H, Litvinchuk A, Chiang AC, et al. (2016) Astrocyte-Microglia Cross Talk through Complement Activation Modulates Amyloid Pathology in Mouse Models of Alzheimer's Disease. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 577-89
Kim JY, Grunke SD, Jankowsky JL. (2016) Widespread Neuronal Transduction of the Rodent CNS via Neonatal Viral Injection. Methods in Molecular Biology (Clifton, N.J.). 1382: 239-50
Wang J, Wegener JE, Huang TW, et al. (2015) Corrigendum: Wild-type microglia do not reverse pathology in mouse models of Rett syndrome. Nature
Liu P, Reed MN, Kotilinek LA, et al. (2015) Quaternary Structure Defines a Large Class of Amyloid-β Oligomers Neutralized by Sequestration. Cell Reports. 11: 1760-71
Wang J, Wegener JE, Huang TW, et al. (2015) Wild-type microglia do not reverse pathology in mouse models of Rett syndrome. Nature. 521: E1-4
Yetman MJ, Lillehaug S, Bjaalie JG, et al. (2015) Transgene expression in the Nop-tTA driver line is not inherently restricted to the entorhinal cortex. Brain Structure & Function
Lian H, Yang L, Cole A, et al. (2015) NFκB-activated astroglial release of complement C3 compromises neuronal morphology and function associated with Alzheimer's disease. Neuron. 85: 101-15
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