Javier H. Jara, Ph.D.
Affiliations: | 2008 | The University of North Dakota, Grand Forks, ND, United States |
Area:
Neuroscience Biology, General, Cell BiologyGoogle:
"Javier Jara"Mean distance: 42746.4
Parents
Sign in to add mentor| Colin K. Combs | grad student | 2008 | University of North Dakota | |
| (Tumor necrosis factor alpha modulates NMDA receptors in the central nervous system.) | ||||
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Publications
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| Genç B, Jara JH, Sanchez SS, et al. (2021) Upper motor neurons are a target for gene therapy and UCHL1 is necessary and sufficient to improve cellular integrity of diseased upper motor neurons. Gene Therapy |
| Jara JH, Sheets PL, Nigro MJ, et al. (2020) The Electrophysiological Determinants of Corticospinal Motor Neuron Vulnerability in ALS. Frontiers in Molecular Neuroscience. 13: 73 |
| Jara JH, Gautam M, Kocak N, et al. (2019) MCP1-CCR2 and neuroinflammation in the ALS motor cortex with TDP-43 pathology. Journal of Neuroinflammation. 16: 196 |
| Gautam M, Jara JH, Kocak N, et al. (2018) Mitochondria, ER, and nuclear membrane defects reveal early mechanisms for upper motor neuron vulnerability with respect to TDP-43 pathology. Acta Neuropathologica |
| Jara JH, Genç B, Stanford MJ, et al. (2017) Evidence for an early innate immune response in the motor cortex of ALS. Journal of Neuroinflammation. 14: 129 |
| Genç B, Jara JH, Lagrimas AK, et al. (2017) Apical dendrite degeneration, a novel cellular pathology for Betz cells in ALS. Scientific Reports. 7: 41765 |
| Genç B, Jara JH, Schultz MC, et al. (2016) Absence of UCHL 1 function leads to selective motor neuropathy. Annals of Clinical and Translational Neurology. 3: 331-45 |
| Gautam M, Jara JH, Sekerkova G, et al. (2016) Absence of alsin function leads to corticospinal motor neuron vulnerability via novel disease mechanisms. Human Molecular Genetics |
| Jara JH, Stanford MJ, Zhu Y, et al. (2015) Healthy and diseased corticospinal motor neurons are selectively transduced upon direct AAV2-2 injection into the motor cortex. Gene Therapy |
| Jara JH, Genç B, Cox GA, et al. (2015) Corticospinal Motor Neurons Are Susceptible to Increased ER Stress and Display Profound Degeneration in the Absence of UCHL1 Function. Cerebral Cortex (New York, N.Y. : 1991) |