James S. Trimmer
Affiliations: | University of California, Davis, Davis, CA |
Area:
Ion channelsWebsite:
http://www.ucdmc.ucdavis.edu/pharmacology/departmentlabs/jimtrimmer.htmlGoogle:
"James Trimmer"Mean distance: 15.31 (cluster 28) | S | N | B | C | P |
Parents
Sign in to add mentor| Victor Vacquier | grad student | Scripps Institute | |
| William S. Agnew | post-doc | Yale School of Medicine (Chemistry Tree) |
Children
Sign in to add trainee| Angie M Michaiel | research assistant | 2009-2012 | UC Davis |
| Jennifer N Jahncke | research assistant | 2013-2014 | UC Davis |
| Robert H. Scannevin | grad student | SUNY Stony Brook | |
| Hannah I. Bishop | grad student | 2010- | UC Davis |
| Louis N. Manganas | grad student | 2001 | SUNY Stony Brook |
| Denise Cook | grad student | 2005-2007 | UC Davis |
| Danielle N. Mandikian | grad student | 2013 | UC Davis |
| Oscar Cerda | post-doc | UC Davis | |
| Matthew N. Rasband | post-doc | SUNY Stony Brook | |
| Hiroaki Misonou | post-doc | 2001-2006 | UC Davis |
BETA: Related publications
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Publications
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| Casas M, Murray KD, Hino K, et al. (2023) NPC1-dependent alterations in K2.1-Ca1.2 nanodomains drive neuronal death in models of Niemann-Pick Type C disease. Nature Communications. 14: 4553 |
| Santana L, Matsumoto C, O'Dwyer S, et al. (2023) The formation of KV2.1 macro-clusters is required for sex-specific differences in L-type CaV1.2 clustering and function in arterial myocytes. Research Square |
| Colecraft HM, Trimmer JS. (2022) Controlling ion channel function with renewable recombinant antibodies. The Journal of Physiology |
| Vierra NC, Trimmer JS. (2022) Ion Channel Partnerships: Odd and Not-So-Odd Couples Controlling Neuronal Ion Channel Function. International Journal of Molecular Sciences. 23 |
| Vierra NC, O'Dwyer SC, Matsumoto C, et al. (2021) Regulation of neuronal excitation-transcription coupling by Kv2.1-induced clustering of somatic L-type Ca channels at ER-PM junctions. Proceedings of the National Academy of Sciences of the United States of America. 118 |
| Fortenbach C, Peinado Allina G, Shores CM, et al. (2021) Loss of the K+ channel Kv2.1 greatly reduces outward dark current and causes ionic dysregulation and degeneration in rod photoreceptors. The Journal of General Physiology. 153 |
| Rivas J, Díaz N, Silva I, et al. (2020) KCTD5, a novel TRPM4-regulatory protein required for cell migration as a new predictor for breast cancer prognosis. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology |
| O'Dwyer SC, Palacio S, Matsumoto C, et al. (2020) Kv2.1 channels play opposing roles in regulating membrane potential, Ca channel function, and myogenic tone in arterial smooth muscle. Proceedings of the National Academy of Sciences of the United States of America. 117: 3858-3866 |
| Maduke M, Sabatini BL, Trimmer JS. (2020) Decision letter: Ca2+ entry through NaV channels generates submillisecond axonal Ca2+ signaling Elife |
| Vierra NC, Kirmiz M, van der List D, et al. (2019) Kv2.1 mediates spatial and functional coupling of L-type calcium channels and ryanodine receptors in mammalian neurons. Elife. 8 |