Dean J. Burkin

Affiliations: 
Cell and Molecular Pharmacology and Physiology University of Nevada, Reno, Reno, NV, United States 
Area:
Molecular Biology, Neuroscience Biology
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"Dean Burkin"
Mean distance: 21373.2
 
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Publications

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Barraza-Flores P, Fontelonga TM, Wuebbles RD, et al. (2019) Laminin-111 protein therapy enhances muscle regeneration and repair in the GRMD dog model of Duchenne muscular dystrophy. Human Molecular Genetics
Fontelonga TM, Jordan B, Nunes AM, et al. (2019) Sunitinib promotes myogenic regeneration and mitigates disease progression in the mdx mouse model of duchenne muscular dystrophy. Human Molecular Genetics
Wuebbles RD, Cruz V, Van Ry P, et al. (2019) Human Galectin-1 Improves Sarcolemma Stability and Muscle Vascularization in the mdx Mouse Model of Duchenne Muscular Dystrophy. Molecular Therapy. Methods & Clinical Development. 13: 145-153
Sarathy A, Nunes AM, Fontelonga TM, et al. (2017) Commentary: SU9516 increases α7β1 Integrin and Ameliorates Disease Progression in the mdx Mouse Model of Duchenne Muscular Dystrophy. Journal of Rare Diseases Research & Treatment. 2: 1-4
Nunes AM, Barraza-Flores P, Smith CR, et al. (2017) Integrin α7: a major driver and therapeutic target for glioblastoma malignancy. Stem Cell Investigation. 4: 97
Van Ry PM, Fontelonga TM, Barraza-Flores P, et al. (2017) ECM-Related Myopathies and Muscular Dystrophies: Pros and Cons of Protein Therapies. Comprehensive Physiology. 7: 1519-1536
Willmann R, Gordish-Dressman H, Meinen S, et al. (2017) Improving Reproducibility of Phenotypic Assessments in the DyW Mouse Model of Laminin-α2 Related Congenital Muscular Dystrophy. Journal of Neuromuscular Diseases
Lewon M, Peters CM, Van Ry PM, et al. (2017) Evaluation of the behavioral characteristics of the mdx mouse model of duchenne muscular dystrophy through operant conditioning procedures. Behavioural Processes. 142: 8-20
Sarathy A, Wuebbles RD, Fontelonga TM, et al. (2017) SU9516 Increases α7β1 Integrin and Ameliorates Disease Progression in the mdx Mouse Model of Duchenne Muscular Dystrophy. Molecular Therapy : the Journal of the American Society of Gene Therapy
Nunes AM, Wuebbles RD, Sarathy A, et al. (2017) Impaired fetal muscle development and JAK-STAT activation mark disease onset and progression in a mouse model for merosin-deficient congenital muscular dystrophy. Human Molecular Genetics
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