Francesco Tamagnini

Affiliations: 
University of Bristol, Bristol, England, United Kingdom 
Area:
Perirhinal cortex, Nitric oxide, endocannabinoids, Alzheimer's disease
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"Francesco Tamagnini"
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Publications

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Bianchi R, Eilers W, Pellati F, et al. (2020) Hippocampal synaptic and membrane function in the DBA/2J-mdx mouse model of Duchenne muscular dystrophy. Molecular and Cellular Neurosciences. 103482
Stephen TL, Tamagnini F, Piegsa J, et al. (2019) Imbalance in the response of pre- and post-synaptic components to amyloidopathy. Scientific Reports. 9: 14837
Palombo F, Masia F, Mattana S, et al. (2018) Hyperspectral analysis applied to micro-Brillouin maps of amyloid-beta plaques in Alzheimer's disease brains. The Analyst
Mattana S, Caponi S, Tamagnini F, et al. (2017) Viscoelasticity of amyloid plaques in transgenic mouse brain studied by Brillouin microspectroscopy and correlative Raman analysis. Journal of Innovative Optical Health Sciences. 10
Tamagnini F, Walsh DA, Brown JT, et al. (2017) Hippocampal neurophysiology is modified by a disease-associated C-terminal fragment of tau protein. Neurobiology of Aging. 60: 44-56
Tamagnini F, Novelia J, Kerrigan TL, et al. (2015) Altered intrinsic excitability of hippocampal CA1 pyramidal neurons in aged PDAPP mice. Frontiers in Cellular Neuroscience. 9: 372
Tamagnini F, Scullion S, Brown JT, et al. (2015) Intrinsic excitability changes induced by acute treatment of hippocampal CA1 pyramidal neurons with exogenous amyloid β peptide. Hippocampus. 25: 786-97
Tamagnini F, Scullion S, Brown JT, et al. (2014) Low concentrations of the solvent dimethyl sulphoxide alter intrinsic excitability properties of cortical and hippocampal pyramidal cells. Plos One. 9: e92557
Tamagnini F, Barker G, Warburton EC, et al. (2013) Nitric oxide-dependent long-term depression but not endocannabinoid-mediated long-term potentiation is crucial for visual recognition memory. The Journal of Physiology. 591: 3963-79
Balietti M, Giorgetti B, Casoli T, et al. (2013) Early selective vulnerability of synapses and synaptic mitochondria in the hippocampal CA1 region of the Tg2576 mouse model of Alzheimer's disease. Journal of Alzheimer's Disease : Jad. 34: 887-96
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