Kathryn E. Medders, Ph.D.
|2010||Molecular Pathology||University of California, San Diego, La Jolla, CA|
Area:Neuroscience Biology, Molecular Biology
Mean distance: 53433
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|Sanchez AB, Medders KE, Maung R, et al. (2016) CXCL12-induced neurotoxicity critically depends on NMDA receptor-gated and L-type Ca(2+) channels upstream of p38 MAPK. Journal of Neuroinflammation. 13: 252|
|Maung R, Hoefer MM, Sanchez AB, et al. (2014) CCR5 knockout prevents neuronal injury and behavioral impairment induced in a transgenic mouse model by a CXCR4-using HIV-1 glycoprotein 120. Journal of Immunology (Baltimore, Md. : 1950). 193: 1895-910|
|Maung R, Medders KE, Sejbuk NE, et al. (2012) Genetic knockouts suggest a critical role for HIV co-receptors in models of HIV gp120-induced brain injury. Journal of Neuroimmune Pharmacology : the Official Journal of the Society On Neuroimmune Pharmacology. 7: 306-18|
|Medders KE, Kaul M. (2011) Mitogen-activated protein kinase p38 in HIV infection and associated brain injury. Journal of Neuroimmune Pharmacology : the Official Journal of the Society On Neuroimmune Pharmacology. 6: 202-15|
|Medders KE, Sejbuk NE, Maung R, et al. (2010) Activation of p38 MAPK is required in monocytic and neuronal cells for HIV glycoprotein 120-induced neurotoxicity. Journal of Immunology (Baltimore, Md. : 1950). 185: 4883-95|
|Kaul M, Ma Q, Medders KE, et al. (2007) HIV-1 coreceptors CCR5 and CXCR4 both mediate neuronal cell death but CCR5 paradoxically can also contribute to protection. Cell Death and Differentiation. 14: 296-305|