Luigi Puglielli
Affiliations: | University of Wisconsin, Madison, Madison, WI |
Area:
Molecular Biology, Cell Biology, Neuroscience BiologyGoogle:
"Luigi Puglielli"Mean distance: 53433
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Publications
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| Murie M, Peng Y, Rigby MJ, et al. (2022) ATase inhibition rescues age-associated proteotoxicity of the secretory pathway. Communications Biology. 5: 173 |
| Sheehan BK, Orefice NS, Peng Y, et al. (2021) ATG9A regulates proteostasis through reticulophagy receptors FAM134B and SEC62 and folding chaperones CALR and HSPB1. Iscience. 24: 102315 |
| Rigby MJ, Lawton AJ, Kaur G, et al. (2021) Endoplasmic reticulum acetyltransferases Atase1 and Atase2 differentially regulate reticulophagy, macroautophagy and cellular acetyl-CoA metabolism. Communications Biology. 4: 454 |
| Dieterich IA, Cui Y, Braun MM, et al. (2021) Acetyl-CoA flux from the cytosol to the ER regulates engagement and quality of the secretory pathway. Scientific Reports. 11: 2013 |
| Cooley MM, Thomas DDH, Deans K, et al. (2020) Deficient Endoplasmic Reticulum Acetyl-CoA Import in Pancreatic Acinar Cells Leads to Chronic Pancreatitis. Cellular and Molecular Gastroenterology and Hepatology |
| Rigby MJ, Ding Y, Farrugia MA, et al. (2020) The endoplasmic reticulum acetyltransferases ATase1/NAT8B and ATase2/NAT8 are differentially regulated to adjust engagement of the secretory pathway. Journal of Neurochemistry. e14958 |
| Dieterich IA, Lawton AJ, Peng Y, et al. (2019) Acetyl-CoA flux regulates the proteome and acetyl-proteome to maintain intracellular metabolic crosstalk. Nature Communications. 10: 3929 |
| Chellappa K, Brinkman JA, Mukherjee S, et al. (2019) Hypothalamic mTORC2 is essential for metabolic health and longevity. Aging Cell. e13014 |
| Farrugia MA, Puglielli L. (2018) Nε-lysine acetylation in the endoplasmic reticulum - a novel cellular mechanism that regulates proteostasis and autophagy. Journal of Cell Science. 131 |
| Lu MH, Ji WL, Xu DE, et al. (2018) Inhibition of sphingomyelin synthase 1 ameliorates alzheimer-like pathology in APP/PS1 transgenic mice through promoting lysosomal degradation of BACE1. Experimental Neurology |