Haesun A. Kim
Affiliations: | Graduate School - Newark | Rutgers The State University of New Jersey - Newark, United States |
Area:
Cell Biology, Neurobiology Biology, Human DevelopmentGoogle:
"Haesun Kim"Mean distance: 35622
Children
Sign in to add traineeDarshan J. Desai | grad student | 2010 | Rutgers, Newark |
Po-Lung Yang | grad student | 2011 | Rutgers, Newark |
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Publications
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Patel AA, Kim H, Ramesh R, et al. (2024) TFEB/3 govern repair Schwann cell generation and function following peripheral nerve injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Adams AA, Wood TL, Kim HA. (2023) Mature and Myelinating Oligodendrocytes Are Specifically Vulnerable to Mild Fluid Percussion Injury in Mice. Neurotrauma Reports. 4: 433-446 |
Adams AA, Li Y, Kim HA, et al. (2023) Dorsal root ganglion neurons recapitulate the traumatic axonal injury of CNS neurons in response to a rapid stretch . Frontiers in Cellular Neuroscience. 17: 1111403 |
Kim J, Adams AA, Gokina P, et al. (2020) Mechanical stretch induces myelin protein loss in oligodendrocytes by activating Erk1/2 in a calcium-dependent manner. Glia |
Kim J, Kim HA. (2018) Isolation and Expansion of Schwann Cells from Transgenic Mouse Models. Methods in Molecular Biology (Clifton, N.J.). 1739: 39-48 |
Kim J, Elias A, Lee T, et al. (2017) Tissue Inhibitor of Metalloproteinase-3 Promotes Schwann Cell Myelination. Asn Neuro. 9: 1759091417745425 |
Basak S, Desai DJ, Rho EH, et al. (2015) E-Cadherin enhances neuregulin signaling and promotes Schwann cell myelination. Glia |
Kim HA, Mindos T, Parkinson DB. (2013) Plastic fantastic: Schwann cells and repair of the peripheral nervous system. Stem Cells Translational Medicine. 2: 553-7 |
Yang DP, Kim J, Syed N, et al. (2012) p38 MAPK activation promotes denervated Schwann cell phenotype and functions as a negative regulator of Schwann cell differentiation and myelination. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 7158-68 |
Chen Y, Wang H, Yoon SO, et al. (2011) HDAC-mediated deacetylation of NF-κB is critical for Schwann cell myelination. Nature Neuroscience. 14: 437-41 |