Matthew T. Rich
Affiliations: | Neuroscience | University of Pittsburgh, Pittsburgh, PA, United States |
Area:
Associative Learning, Drug-Induced Memory FormationGoogle:
"Matthew Rich"Mean distance: 16.78 (cluster 19) | S | N | B | C | P |
Parents
Sign in to add mentor| Srdjan D. Antic | research assistant | University of Connecticut Health Center | |
| Mary M. Torregrossa | grad student | 2013- | University of Pittsburgh |
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Publications
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| Rich MT, Worobey SJ, Mankame S, et al. (2023) Sex-dependent fear memory impairment in cocaine-sired rat offspring. Science Advances. 9: eadf6039 |
| Pierce RC, Rich MT, Swinford-Jackson SE. (2023) Addiction neuroscience goes nuclear: A role for the transcription factor RXRα. Neuron. 111: 1351-1353 |
| Swinford-Jackson SE, Huffman PJ, Knouse MC, et al. (2022) High frequency DBS-like optogenetic stimulation of nucleus accumbens dopamine D2 receptor-containing neurons attenuates cocaine reinstatement in male rats. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology |
| Rich MT, Huang YH, Torregrossa MM. (2021) Using Optogenetics to Reverse Neuroplasticity and Inhibit Cocaine Seeking in Rats. Journal of Visualized Experiments : Jove |
| Hernandez NS, Weir VR, Ragnini K, et al. (2020) GLP-1 receptor signaling in the laterodorsal tegmental nucleus attenuates cocaine seeking by activating GABAergic circuits that project to the VTA. Molecular Psychiatry |
| Rich MT, Huang YH, Torregrossa MM. (2019) Calcineurin Promotes Neuroplastic Changes in the Amygdala Associated with Weakened Cocaine-Cue Memories. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Rich MT, Huang YH, Torregrossa MM. (2019) Plasticity at Thalamo-amygdala Synapses Regulates Cocaine-Cue Memory Formation and Extinction. Cell Reports. 26: 1010-1020.e5 |
| Rich MT, Torregrossa MM. (2017) Molecular and Synaptic Mechanisms Regulating Drug-Associated Memories: Towards a Bidirectional Treatment Strategy. Brain Research Bulletin |
| Rich MT, Cahanap T, Huang Y, et al. (2017) Cocaine-cue memory extinction is associated with depotentiation at amygdala synapses Drug and Alcohol Dependence. 171: e176 |
| Rich MT, Abbott TB, Chung L, et al. (2016) Phosphoproteomic Analysis Reveals a Novel Mechanism of CaMKIIα Regulation Inversely Induced by Cocaine Memory Extinction versus Reconsolidation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 7613-27 |