David Pleasure
Affiliations: | Comparative Pathology | University of California, Davis, Davis, CA |
Area:
Pathology, Immunology, Neuroscience BiologyGoogle:
"David Pleasure"Mean distance: 53433
Children
Sign in to add trainee| Vanessa L. Hull | grad student | 2019- | UC Davis |
| Emily Mills Ko | grad student | 2014 | UC Davis |
| Judith B Grinspan | post-doc | 1984-1989 | Children's Hospital of Philadelphia/UPenn |
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Publications
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| Hull VL, Wang Y, McDonough J, et al. (2024) Astroglial conditional Slc13a3 knockout is therapeutic in murine Canavan leukodystrophy. Annals of Clinical and Translational Neurology |
| Hull VL, Wang Y, Burns T, et al. (2023) Pathological Bergmann glia alterations and disrupted calcium dynamics in ataxic Canavan disease mice. Glia |
| Mayrhofer F, Dariychuk Z, Zhen A, et al. (2021) Reduction in CD11c microglia correlates with clinical progression in chronic experimental autoimmune demyelination. Neurobiology of Disease. 105556 |
| Wang Y, Hull V, Sternbach S, et al. (2021) Ablating the transporter NaDC3 prevents leukodystrophy in Canavan disease mice. Annals of Neurology |
| Hull V, Wang Y, Burns T, et al. (2020) Antisense oligonucleotide reverses leukodystrophy in Canavan disease mice. Annals of Neurology |
| Wang YZ, Fan H, Ji Y, et al. (2019) Olig2 regulates terminal differentiation and maturation of peripheral olfactory sensory neurons. Cellular and Molecular Life Sciences : Cmls |
| Pleasure D, Guo F, Chechneva O, et al. (2018) Pathophysiology and Treatment of Canavan Disease. Neurochemical Research |
| Horiuchi M, Suzuki-Horiuchi Y, Akiyama T, et al. (2017) Differing intrinsic biological properties between forebrain and spinal oligodendroglial lineage cells. Journal of Neurochemistry |
| Moreno MA, Burns T, Yao P, et al. (2016) Therapeutic depletion of monocyte-derived cells protects from long-term axonal loss in experimental autoimmune encephalomyelitis. Journal of Neuroimmunology. 290: 36-46 |
| Chung SH, Biswas S, Selvaraj V, et al. (2015) The p38α mitogen-activated protein kinase is a key regulator of myelination and remyelination in the CNS. Cell Death & Disease. 6: e1748 |