Markus Glatzel, M.D.

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Wasilewski D, Villalba-Moreno ND, Stange I, et al. (2022) Reactive Astrocytes Contribute to Alzheimer's Disease-Related Neurotoxicity and Synaptotoxicity in a Neuron-Astrocyte Co-culture Assay. Frontiers in Cellular Neuroscience. 15: 739411
Krasemann S, Haferkamp U, Pfefferle S, et al. (2022) The blood-brain barrier is dysregulated in COVID-19 and serves as a CNS entry route for SARS-CoV-2. Stem Cell Reports
O'Hare M, Amarnani D, Whitmore HAB, et al. (2021) Targeting RUNX1 prevents pulmonary fibrosis and reduces expression of SARS-CoV-2 host mediators. The American Journal of Pathology
Sepulveda-Falla D, Chavez-Gutierrez L, Portelius E, et al. (2020) A multifactorial model of pathology for age of onset heterogeneity in familial Alzheimer's disease. Acta Neuropathologica
Dinkel F, Trujillo-Rodriguez D, Villegas A, et al. (2020) Decreased Deposition of Beta-Amyloid 1-38 and Increased Deposition of Beta-Amyloid 1-42 in Brain Tissue of Presenilin-1 E280A Familial Alzheimer's Disease Patients. Frontiers in Aging Neuroscience. 12: 220
Callender JA, Sevillano AM, Soldau K, et al. (2020) Prion protein post-translational modifications modulate heparan sulfate binding and limit aggregate size in prion disease. Neurobiology of Disease. 104955
Mohammadi B, Linsenmeier L, Shafiq M, et al. (2020) Transgenic Overexpression of the Disordered Prion Protein N1 Fragment in Mice Does Not Protect Against Neurodegenerative Diseases Due to Impaired ER Translocation. Molecular Neurobiology
Sabogal-Guáqueta AM, Arias-Londoño JD, Gutierrez-Vargas J, et al. (2020) Common disbalance in the brain parenchyma of dementias: Phospholipid profile analysis between CADASIL and sporadic Alzheimer's disease. Biochimica Et Biophysica Acta. Molecular Basis of Disease. 1866: 165797
Rojas-Charry L, Calero-Martinez S, Morganti C, et al. (2020) Susceptibility to cellular stress in PS1 mutant N2a cells is associated with mitochondrial defects and altered calcium homeostasis. Scientific Reports. 10: 6455
Sevillano AM, Aguilar-Calvo P, Kurt TD, et al. (2020) Prion protein glycans reduce intracerebral fibril formation and spongiosis in prion disease. The Journal of Clinical Investigation