William C. Wetsel, Ph.D.

Affiliations: 
Neurobiology Duke University, Durham, NC 
Area:
Behavioral neuroscience
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"William Wetsel"
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G. Mark Freeman, Jr research assistant 2000-2003 Duke
Derrick L. Nehrenberg grad student 2004 UNC Chapel Hill
Meng Chen research scientist 2010-2012 Duke
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Publications

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Martini ML, Ray C, Yu X, et al. (2019) Designing Functionally Selective Non-Catechol Dopamine D1 Receptor Agonists with Potent In Vivo Antiparkinsonian Activity. Acs Chemical Neuroscience
Pogorelov VM, Kao HT, Augustine GJ, et al. (2019) Postsynaptic Mechanisms Render Syn I/II/III Mice Highly Responsive to Psychostimulants. The International Journal of Neuropsychopharmacology
Shen Y, McCorvy JD, Martini ML, et al. (2019) D2 Dopamine Receptor G protein-biased Partial Agonists Based on Cariprazine. Journal of Medicinal Chemistry
McGaughey KD, Yilmaz-Swenson T, Elsayed NM, et al. (2019) Relative abundance of Akkermansia spp. and other bacterial phylotypes correlates with anxiety- and depressive-like behavior following social defeat in mice. Scientific Reports. 9: 3281
Arbogast T, Razaz P, Ellegood J, et al. (2018) Kctd13-deficient mice display short-term memory impairment and sex-dependent genetic interactions. Human Molecular Genetics
Huffman WJ, Subramaniyan S, Rodriguiz RM, et al. (2018) Modulation of neuroinflammation and memory dysfunction using percutaneous vagus nerve stimulation in mice. Brain Stimulation
Bey AL, Wang X, Yan H, et al. (2018) Brain region-specific disruption of Shank3 in mice reveals a dissociation for cortical and striatal circuits in autism-related behaviors. Translational Psychiatry. 8: 94
Martyn AC, Toth K, Schmalzigaug R, et al. (2018) GIT1 regulates synaptic structural plasticity underlying learning. Plos One. 13: e0194350
Pappas AL, Bey AL, Wang X, et al. (2017) Deficiency of Shank2 causes mania-like behavior that responds to mood stabilizers. Jci Insight. 2
Butler RK, Ehling S, Barbar M, et al. (2017) Distinct neuronal populations in the basolateral and central amygdala are activated with acute pain, conditioned fear, and fear-conditioned analgesia. Neuroscience Letters
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