Bruce Winegar

Anesthesia Stanford University Medical Center, Palo Alto, CA, United States 
"Bruce Winegar"
Mean distance: 16.06 (cluster 11)
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Winegar BD, MacIver MB. (2006) Isoflurane depresses hippocampal CA1 glutamate nerve terminals without inhibiting fiber volleys. Bmc Neuroscience. 7: 5
Gerstin KM, Gong DH, Abdallah M, et al. (2003) Mutation of KCNK5 or Kir3.2 potassium channels in mice does not change minimum alveolar anesthetic concentration. Anesthesia and Analgesia. 96: 1345-9, table of con
Shin WJ, Winegar BD. (2003) Modulation of noninactivating K+ channels in rat cerebellar granule neurons by halothane, isoflurane, and sevoflurane. Anesthesia and Analgesia. 96: 1340-4, table of con
Kindler CH, Paul M, Zou H, et al. (2003) Amide local anesthetics potently inhibit the human tandem pore domain background K+ channel TASK-2 (KCNK5). The Journal of Pharmacology and Experimental Therapeutics. 306: 84-92
Gabriel A, Abdallah M, Yost CS, et al. (2002) Localization of the tandem pore domain K+ channel KCNK5 (TASK-2) in the rat central nervous system. Brain Research. Molecular Brain Research. 98: 153-63
Gray AT, Zhao BB, Kindler CH, et al. (2000) Volatile anesthetics activate the human tandem pore domain baseline K+ channel KCNK5. Anesthesiology. 92: 1722-30
Yost CS, Gray AT, Winegar BD, et al. (1998) Baseline K+ channels as targets of general anesthetics: studies of the action of volatile anesthetics on TOK1. Toxicology Letters. 100: 293-300
Winegar BD, Yost CS. (1998) Activation of single potassium channels in rat cerebellar granule cells by volatile anesthetics. Toxicology Letters. 100: 287-91
Winegar BD, Yost CS. (1998) Volatile anesthetics directly activate baseline S K+ channels in aplysia neurons. Brain Research. 807: 255-62
Gray AT, Winegar BD, Leonoudakis DJ, et al. (1998) TOK1 is a volatile anesthetic stimulated K+ channel. Anesthesiology. 88: 1076-84
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