Joshua Goldhaber
Affiliations: | Molec, Cell, & Integ Physiology 0568 | University of California, Los Angeles, Los Angeles, CA |
Area:
Physiology Biology, Neuroscience BiologyGoogle:
"Joshua Goldhaber"Mean distance: 35622
Children
Sign in to add traineeOlujimi A. Ajijola | grad student | 2013 | UCLA |
Jessica M. Gluck | grad student | 2013 | UCLA |
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Publications
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Scranton K, John S, Angelini M, et al. (2024) Cardiac function is regulated by the sodium-dependent inhibition of the sodium-calcium exchanger NCX1. Nature Communications. 15: 3831 |
Bögeholz N, Knappe V, Pauls P, et al. (2022) Increased perpetuation of whole-heart ventricular arrhythmia in heterozygous Na/Ca exchanger knockout mice. International Journal of Cardiology. Heart & Vasculature. 44: 101168 |
Yue X, Hazan A, Lotteau S, et al. (2020) Na/Ca exchange in the atrium: Role in sinoatrial node pacemaking and excitation-contraction coupling. Cell Calcium. 87: 102167 |
Yoshie K, Rajendran PS, Massoud L, et al. (2019) Cardiac TRPV1-afferent signaling promotes arrhythmogenic ventricular remodeling after myocardial infarction. Jci Insight |
Liang W, Han P, Kim EH, et al. (2019) Canonical Wnt signaling promotes pacemaker cell specification of cardiac mesodermal cells derived from mouse and human embryonic stem cells. Stem Cells (Dayton, Ohio) |
Aminzadeh MA, Rogers RG, Fournier M, et al. (2018) Exosome-Mediated Benefits of Cell Therapy in Mouse and Human Models of Duchenne Muscular Dystrophy. Stem Cell Reports |
John S, Kim B, Olcese R, et al. (2018) Molecular determinants of pH regulation in the cardiac Na+-Ca2+ exchanger. The Journal of General Physiology |
Shattock MJ, Ottolia M, Bers DM, et al. (2015) Na+/Ca2+ exchange and Na+/K+-ATPase in the heart. The Journal of Physiology. 593: 1361-82 |
Anderson ME, Goldhaber J, Houser SR, et al. (2014) Embryonic stem cell-derived cardiac myocytes are not ready for human trials. Circulation Research. 115: 335-8 |
Groenke S, Larson ED, Alber S, et al. (2013) Complete atrial-specific knockout of sodium-calcium exchange eliminates sinoatrial node pacemaker activity. Plos One. 8: e81633 |