Chenjian Li

Affiliations: 
Weill Cornell Medical College, New York, NY, United States 
Area:
Neuroscience Biology, Genetics
Google:
"Chenjian Li"
Mean distance: 35622
 
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Zhi L, Zhao J, Jaffe D, et al. (2022) Measurement of Oxygen Consumption Rate in Acute Striatal Slices from Adult Mice. Journal of Visualized Experiments : Jove
Shenoy SA, Zheng S, Liu W, et al. (2022) A novel and accurate full-length HTT mouse model for Huntington's disease. Elife. 11
Yang X, Bayat V, DiDonato N, et al. (2019) Genetic and genomic studies of pathogenic EXOSC2 mutations in the newly described disease SHRF implicate the autophagy pathway in disease pathogenesis. Human Molecular Genetics
Zhi L, Qin Q, Muqeem T, et al. (2018) Loss of PINK1 causes age-dependent decrease of dopamine release and mitochondrial dysfunction. Neurobiology of Aging. 75: 1-10
Chen BB, Coon TA, Glasser JR, et al. (2014) E3 ligase subunit Fbxo15 and PINK1 kinase regulate cardiolipin synthase 1 stability and mitochondrial function in pneumonia. Cell Reports. 7: 476-87
Cannon JR, Geghman KD, Tapias V, et al. (2013) Expression of human E46K-mutated α-synuclein in BAC-transgenic rats replicates early-stage Parkinson's disease features and enhances vulnerability to mitochondrial impairment. Experimental Neurology. 240: 44-56
Zheng S, Geghman K, Shenoy S, et al. (2012) Retake the center stage--new development of rat genetics. Journal of Genetics and Genomics = Yi Chuan Xue Bao. 39: 261-8
Huang Y, Shenoy S, Lu B, et al. (2012) Kinase signaling dysfunction in Parkinson's disease: a reverse genetic approach in Drosophila. Journal of Neurogenetics. 26: 158-67
Xu Q, Shenoy S, Li C. (2012) Mouse models for LRRK2 Parkinson's disease. Parkinsonism & Related Disorders. 18: S186-9
Geghman K, Li C. (2011) Practical considerations of genetic rodent models for neurodegenerative diseases. Methods in Molecular Biology (Clifton, N.J.). 793: 185-93
See more...