Huizhong Xiong, Ph.D.
Affiliations: | 2012 | Basic Medical Science | New York University, New York, NY, United States |
Area:
Immunology, Pathology, OncologyGoogle:
"Huizhong Xiong"Mean distance: 30533.1
Parents
Sign in to add mentor| Juan J. Lafaille | grad student | 2012 | NYU | |
| (Pathogenesis of anaphylaxis and T cell acute lymphoblastic leukemia (T-ALL).) | ||||
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Publications
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| Xiong H, Mancini M, Gobert M, et al. (2021) Spleen plays a major role in DLL4-driven acute T-cell lymphoblastic leukemia. Theranostics. 11: 1594-1608 |
| Xiong H, Mittman S, Rodriguez R, et al. (2019) Coexpression of Inhibitory Receptors Enriches for Activated and Functional CD8 T Cells in Murine Syngeneic Tumor Models. Cancer Immunology Research |
| Xiong H, Mittman S, Rodriguez R, et al. (2019) Anti-PD-L1 treatment results in functional remodeling of the macrophage compartment. Cancer Research |
| Xiong H, Mittman S, Rodriguez R, et al. (2019) Abstract A120: Intratumoral dendritic cell dynamics in responsive and nonresponsive syngeneic murine tumor models Cancer Immunology Research. 7 |
| Xiong H, Moskalenko M, Rodriguez R, et al. (2018) Abstract A31: Reprogramming of tumor-associated macrophages by anti-Program Death Ligand 1 (PDL1) Cancer Immunology Research. 6 |
| Bakst RL, Xiong H, Chen CH, et al. (2017) Inflammatory monocytes promote perineural invasion via CCL2-mediated recruitment and cathepsin B expression. Cancer Research |
| Gallegos AM, Xiong H, Leiner IM, et al. (2016) Control of T cell antigen reactivity via programmed TCR downregulation. Nature Immunology |
| Abt MC, Lewis BB, Caballero S, et al. (2015) Innate Immune Defenses Mediated by Two ILC Subsets Are Critical for Protection against Acute Clostridium difficile Infection. Cell Host & Microbe. 18: 27-37 |
| Xiong H, Pamer EG. (2015) Monocytes and infection: modulator, messenger and effector. Immunobiology. 220: 210-4 |
| Bakst R, Xiong H, Chen C, et al. (2015) Abstract 3167: CCL2 recruits inflammatory monocytes to facilitate perineural invasion Cancer Research. 75: 3167-3167 |