Utpal B. Pajvani, Ph.D.
Affiliations: | 2005 | Yeshiva University, New York, NY, United States |
Area:
Cell Biology, Animal Physiology Biology, Molecular BiologyGoogle:
"Utpal Pajvani"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Philipp E. Scherer | grad student | 2005 | Yeshiva University | |
| (Functional consequences of adiponectin oligomerization state: The role of adiponectin in maintaining systemic insulin sensitivity.) | ||||
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Publications
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| Pajvani UB, Shawber CJ, Samuel VT, et al. (2023) Author Correction: Inhibition of Notch signaling ameliorates insulin resistance in a FoxO1-dependent manner. Nature Medicine |
| Cook JR, Hawkins MA, Pajvani UB. (2023) Liver insulinization as a driver of triglyceride dysmetabolism. Nature Metabolism. 5: 1101-1110 |
| Aaron N, Zahr T, He Y, et al. (2022) Acetylation of PPARγ in macrophages promotes visceral fat degeneration in obesity. Life Metabolism. 1: 258-269 |
| Oh AR, Jeong Y, Yu J, et al. (2022) Hepatocyte Kctd17 inhibition ameliorates glucose intolerance and hepatic steatosis caused by obesity-induced Chrebp stabilization. Gastroenterology |
| He Y, B'nai Taub A, Yu L, et al. (2022) PPARγ Acetylation Orchestrates Adipose Plasticity and Metabolic Rhythms. Advanced Science (Weinheim, Baden-Wurttemberg, Germany). e2204190 |
| Wang L, Yu J, Zhou Q, et al. (2021) TOX4, an insulin receptor-independent regulator of hepatic glucose production, is activated in diabetic liver. Cell Metabolism |
| Kim K, Kang JK, Jung YH, et al. (2021) Adipocyte PHLPP2 inhibition prevents obesity-induced fatty liver. Nature Communications. 12: 1822 |
| Richter LR, Wan Q, Wen D, et al. (2020) Targeted Delivery of Notch Inhibitor Attenuates Obesity-Induced Glucose Intolerance and Liver Fibrosis. Acs Nano |
| BARTOLOME A, PAJVANI U. (2019) 109-OR: Notch Signaling Controls Islet Architecture and ß-Cell Function in the Developed Pancreas Diabetes. 68: 109-OR |
| Kim K, Goldberg IJ, Graham MJ, et al. (2018) γ-Secretase Inhibition Lowers Plasma Triglyceride-Rich Lipoproteins by Stabilizing the LDL Receptor. Cell Metabolism |