Markus Britschgi, Ph.D.
Affiliations: | Roche, Indianapolis, IN, United States |
Area:
Neuroimmunology, neurodegeneration, agingGoogle:
"Markus Britschgi"Mean distance: 16.16 (cluster 32) | S | N | B | C | P |
Parents
Sign in to add mentorTony Wyss-Coray | post-doc | 2003-2009 | Stanford Medical School/Palo Alto VA |
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Publications
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Grathwohl S, Quansah E, Maroof N, et al. (2021) Specific immune modulation of experimental colitis drives enteric alpha-synuclein accumulation and triggers age-related Parkinson-like brain pathology. Free Neuropathology. 2 |
Brigger D, Riether C, van Brummelen R, et al. (2020) Eosinophils regulate adipose tissue inflammation and sustain physical and immunological fitness in old age. Nature Metabolism |
Brüll M, Spreng AS, Gutbier S, et al. (2020) Incorporation of stem cell-derived astrocytes into neuronal organoids to allow neuro-glial interactions in toxicological studies. Altex |
Shahmoradian SH, Lewis AJ, Genoud C, et al. (2019) Lewy pathology in Parkinson's disease consists of crowded organelles and lipid membranes. Nature Neuroscience. 22: 1099-1109 |
Navarro PP, Genoud C, Castaño-Díez D, et al. (2018) Cerebral Corpora amylacea are dense membranous labyrinths containing structurally preserved cell organelles. Scientific Reports. 8: 18046 |
Guerrero-Ferreira R, Taylor NMI, Mona D, et al. (2018) Cryo-EM structure of alpha-synuclein fibrils. Elife. 7 |
Czirr E, Castello NA, Mosher KI, et al. (2017) Microglial complement receptor 3 regulates brain Aβ levels through secreted proteolytic activity. The Journal of Experimental Medicine |
Jaeger PA, Lucin KM, Britschgi M, et al. (2016) Erratum to: Network-driven plasma proteomics expose molecular changes in the Alzheimer's brain. Molecular Neurodegeneration. 11: 42 |
Jaeger PA, Lucin KM, Britschgi M, et al. (2016) Network-driven plasma proteomics expose molecular changes in the Alzheimer's brain. Molecular Neurodegeneration. 11: 31 |
Lehallier B, Essioux L, Gayan J, et al. (2015) Combined Plasma and Cerebrospinal Fluid Signature for the Prediction of Midterm Progression From Mild Cognitive Impairment to Alzheimer Disease. Jama Neurology. 1-10 |