Michael Sendtner
Affiliations: | Institut für Klinische Neurobiologie | Julius-Maximilans-Universität Würzburg, Würzburg, Bayern, Germany |
Area:
Nervous system diseases, synaptic plasticityWebsite:
http://gs.zv.uni-wuerzburg.de/Google:
"Michael Sendtner"Mean distance: 106866
Collaborators
Sign in to add collaborator| Georg W. Kreutzberg | collaborator | Max Planck Institute of Neurobiology |
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Publications
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| Brenner D, Rosenbohm A, Yilmaz R, et al. (2019) Reply: Adult-onset distal spinal muscular atrophy: a new phenotype associated with KIF5A mutations. Brain : a Journal of Neurology |
| Müller K, Brenner D, Weydt P, et al. (2018) Comprehensive analysis of the mutation spectrum in 301 German ALS families. Journal of Neurology, Neurosurgery, and Psychiatry |
| Brenner D, Yilmaz R, Müller K, et al. (2018) Hot-spot KIF5A mutations cause familial ALS. Brain : a Journal of Neurology |
| Lüningschrör P, Binotti B, Dombert B, et al. (2017) Plekhg5-regulated autophagy of synaptic vesicles reveals a pathogenic mechanism in motoneuron disease. Nature Communications. 8: 678 |
| Sivadasan R, Hornburg D, Drepper C, et al. (2016) C9ORF72 interaction with cofilin modulates actin dynamics in motor neurons. Nature Neuroscience |
| Kenna KP, van Doormaal PT, Dekker AM, et al. (2016) NEK1 variants confer susceptibility to amyotrophic lateral sclerosis. Nature Genetics |
| Yadav P, Selvaraj BT, Bender FL, et al. (2016) Neurofilament depletion improves microtubule dynamics via modulation of Stat3/stathmin signaling. Acta Neuropathologica |
| Simon CM, Rauskolb S, Gunnersen JM, et al. (2015) Dysregulated IGFBP5 expression causes axon degeneration and motoneuron loss in diabetic neuropathy. Acta Neuropathologica |
| Wirth B, Barkats M, Martinat C, et al. (2015) Moving towards treatments for spinal muscular atrophy: hopes and limits. Expert Opinion On Emerging Drugs. 1-4 |
| Hoffmann CJ, Harms U, Rex A, et al. (2015) Vascular signal transducer and activator of transcription-3 promotes angiogenesis and neuroplasticity long-term after stroke. Circulation. 131: 1772-82 |