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Samuel Young, Phd

Affiliations: 
Max Planck Florida Institute, Jupiter, FL, United States 
Area:
Molecular mechanism of synaptic function
Website:
http://www.samuelyounglab.org/
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"Samuel Young"
Bio:

Dr. Samuel M. Young, Jr. was most recently an Internal Research Group Leader (2008-2009) in the Department of Membrane Biophysics at the Max Planck Institute for Biophysical Chemistry in Goettingen, Germany.

He conducted his postdoctoral work at the University of North Carolina-Chapel Hill (2000) in the Gene Therapy Center, at the Howard Hughes Medical Institute and the Salk Institute in La Jolla, California (2000-2004) in the Molecular Neurobiology Laboratories, and at the Max Planck Institute for Biochemistry in the Department of Membrane Biophysics (2004-2008).

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Mean distance: 12.91 (cluster 11)
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Parents

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Erwin Neher post-doc Max Planck Florida Institute
Charles F. Stevens post-doc Max Planck Florida Institute

Children

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Zuxin Chen grad student Max Planck Florida Institute
Brati Das grad student Max Planck Florida Institute
Priyanka Prakash Kakad grad student Max Planck Florida Institute
Monica Montesinos post-doc Max Planck Florida Institute
Christian Keine post-doc 2016- Max Planck Florida Institute for Neuroscience
Wei Dong post-doc 2013-2017 Max Planck Florida Institute

Collaborators

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Yukihiro Nakamura collaborator Institut Pasteur
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Publications

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Radulovic T, Dong W, Goral RO, et al. (2020) Presynaptic development is controlled by the core active zone proteins CAST/ELKS. The Journal of Physiology
Thomas CI, Keine C, Okayama S, et al. (2019) Presynaptic mitochondria volume and abundance increase during development of a high-fidelity synapse. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Dong W, Xu M, Radulovic T, et al. (2019) CAST/ELKS regulates presynaptic morphology and calcium channel levels in a developing central synapse Ibro Reports. 6: S145
Lübbert M, Goral RO, Keine C, et al. (2018) Ca2.1 α Subunit Expression Regulates Presynaptic Ca2.1 Abundance and Synaptic Strength at a Central Synapse. Neuron
Dong W, Radulovic T, Goral RO, et al. (2018) CAST/ELKS Proteins Control Voltage-Gated Ca Channel Density and Synaptic Release Probability at a Mammalian Central Synapse. Cell Reports. 24: 284-293.e6
Durán E, Montes MÁ, Jemal I, et al. (2018) Synaptotagmin-7 controls the size of the reserve and resting pools of synaptic vesicles in hippocampal neurons. Cell Calcium. 74: 53-60
Chen C, Satterfield R, Young SM, et al. (2017) Triple Function of Synaptotagmin 7 Ensures Efficiency of High-Frequency Transmission at Central GABAergic Synapses. Cell Reports. 21: 2082-2089
Chen C, Arai I, Satterfield R, et al. (2017) Synaptotagmin 2 Is the Fast Ca(2+) Sensor at a Central Inhibitory Synapse. Cell Reports. 18: 723-736
Montesinos MS, Satterfield R, Young SM. (2016) Helper-Dependent Adenoviral Vectors and Their Use for Neuroscience Applications. Methods in Molecular Biology (Clifton, N.J.). 1474: 73-90
Montesinos MS, Dong W, Goff K, et al. (2015) Presynaptic Deletion of GIT Proteins Results in Increased Synaptic Strength at a Mammalian Central Synapse. Neuron. 88: 918-25
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