Naeem K. Patil, Ph.D.
Affiliations: | 2014 | Interdisciplinary Toxicology | University of Arkansas for Medical Sciences, United States |
Area:
Pharmacology, Medicine and Surgery, GeneralGoogle:
"Naeem Patil"Parents
Sign in to add mentorLee A. MacMillan-Crow | grad student | 2014 | UAMS | |
(Renal mitochondrial dysfunction: Novel therapeutic targets to treat sepsis induced acute kidney injury.) |
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Publications
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Hernandez A, Patil NK, Brewer M, et al. (2023) Pretreatment with a Novel Toll-like Receptor 4 Agonist Phosphorylated Hexaacyl Disaccharide Attenuates Renal Ischemia-Reperfusion Injury. American Journal of Physiology. Renal Physiology |
McBride MA, Owen AM, Stothers CL, et al. (2020) The Metabolic Basis of Immune Dysfunction Following Sepsis and Trauma. Frontiers in Immunology. 11: 1043 |
Hernandez A, Patil NK, Stothers CL, et al. (2019) Immunobiology and Application of Toll-like Receptor 4 Agonists to Augment Host Resistance to Infection. Pharmacological Research. 104502 |
Fukuda S, Ihara K, Bohannon JK, et al. (2019) Monophosphoryl Lipid A Attenuates Multiorgan Dysfunction During Post-Burn Pseudomonas Aeruginosa Pneumonia In Sheep. Shock (Augusta, Ga.) |
Patil NK, Bohannon JK, Hernandez A, et al. (2019) Regulation of leukocyte function by citric acid cycle intermediates. Journal of Leukocyte Biology |
Fensterheim BA, Young JD, Luan L, et al. (2018) The TLR4 Agonist Monophosphoryl Lipid A Drives Broad Resistance to Infection via Dynamic Reprogramming of Macrophage Metabolism. Journal of Immunology (Baltimore, Md. : 1950) |
Patil NK, Luan L, Bohannon JK, et al. (2018) Frontline Science: Anti-PD-L1 protects against infection with common bacterial pathogens after burn injury. Journal of Leukocyte Biology. 103: 23-33 |
Patil NK, Guo Y, Luan L, et al. (2017) Targeting Immune Cell Checkpoints during Sepsis. International Journal of Molecular Sciences. 18 |
Guo Y, Patil NK, Luan L, et al. (2017) The Biology of Natural Killer Cells during Sepsis. Immunology |
Patil NK, Bohannon JK, Sherwood ER. (2016) Immunotherapy: A Promising Approach to Reverse Sepsis-Induced Immunosuppression. Pharmacological Research |