Monica Y. Lee, Ph.D.
Affiliations: | 2011 | University of Virginia, Charlottesville, VA |
Area:
Molecular BiologyGoogle:
"Monica Lee"Parents
Sign in to add mentor| Brian R. Wamhoff | grad student | 2011 | UVA | |
| (Nuclear Factor of Activated T-Cells (NFAT)-mediated Regulation of Vascular Smooth Muscle Cell Phenotypic Modulation.) | ||||
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Publications
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| Hyun J, Lee M, Rehman J, et al. (2021) Notch1 promotes revascularization through modulation of downstream vascular patterning signaling. The Journal of Physiology |
| Lee MY, Gamez-Mendez A, Zhang J, et al. (2018) Endothelial Cell Autonomous Role of Akt1: Regulation of Vascular Tone and Ischemia-Induced Arteriogenesis. Arteriosclerosis, Thrombosis, and Vascular Biology |
| Kuo A, Lee MY, Yang K, et al. (2017) Caveolin-1 regulates lipid droplet metabolism in endothelial cells via autocrine prostacyclin stimulated cAMP-mediated lipolysis. The Journal of Biological Chemistry |
| Jin Y, Xie Y, Ostriker AC, et al. (2017) Opposing Actions of AKT (Protein Kinase B) Isoforms in Vascular Smooth Muscle Injury and Therapeutic Response. Arteriosclerosis, Thrombosis, and Vascular Biology |
| Kuo A, Lee MY, Sessa WC. (2017) Lipid Droplet Biogenesis and Function in the Endothelium. Circulation Research |
| Ola R, Dubrac A, Han J, et al. (2016) PI3 kinase inhibition improves vascular malformations in mouse models of hereditary haemorrhagic telangiectasia. Nature Communications. 7: 13650 |
| Kraehling JR, Chidlow JH, Rajagopal C, et al. (2016) Genome-wide RNAi screen reveals ALK1 mediates LDL uptake and transcytosis in endothelial cells. Nature Communications. 7: 13516 |
| Kraehling JR, Hao Z, Lee MY, et al. (2015) Uncoupling Caveolae from Intracellular Signaling In Vivo. Circulation Research |
| Chamorro-Jorganes A, Lee MY, Araldi E, et al. (2015) VEGF-Induced Expression of miR-17~92 Cluster in Endothelial Cells is Mediated by ERK/ELK1 Activation and Regulates Angiogenesis. Circulation Research |
| Bancroft T, Bouaouina M, Roberts S, et al. (2015) Up-regulation of thrombospondin-2 in Akt1-null mice contributes to compromised tissue repair due to abnormalities in fibroblast function. The Journal of Biological Chemistry. 290: 409-22 |