Isabel Martinez-Garay

Affiliations: 
University of Oxford, Oxford, United Kingdom 
Area:
Corticogenesis, migration
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"Isabel Martinez-Garay"
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Publications

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Galindo-Riera N, Newbold SA, Sledziowska M, et al. (2021) Cellular and behavioral characterization of Pcdh19 mutant mice: subtle molecular changes, increased exploratory behavior and an impact of social environment. Eneuro
Galindo-Riera N, Newbold SA, Sledziowska M, et al. (2021) Cellular and behavioral characterization of Pcdh19 mutant mice: subtle molecular changes, increased exploratory behavior and an impact of social environment. Eneuro
Paramo B, Bachmann SO, Baudouin SJ, et al. (2021) Neuregulin-4 is required for maintaining soma size of pyramidal neurons in the motor cortex. Eneuro
Martinez-Garay I. (2020) Molecular Mechanisms of Cadherin Function During Cortical Migration. Frontiers in Cell and Developmental Biology. 8: 588152
Rueda-Alaña E, Martínez-Garay I, Encinas JM, et al. (2018) Dbx1-Derived Pyramidal Neurons Are Generated Locally in the Developing Murine Neocortex. Frontiers in Neuroscience. 12: 792
Guidi LG, Velayos-Baeza A, Martinez-Garay I, et al. (2018) The Neuronal Migration Hypothesis of Dyslexia: A Critical Evaluation Thirty Years On. The European Journal of Neuroscience
Guidi LG, Mattley J, Martinez-Garay I, et al. (2017) Knockout Mice for Dyslexia Susceptibility Gene Homologs KIAA0319 and KIAA0319L have Unaffected Neuronal Migration but Display Abnormal Auditory Processing. Cerebral Cortex (New York, N.Y. : 1991). 1-15
Diggle CP, Martinez-Garay I, Molnar Z, et al. (2017) A tubulin alpha 8 mouse knockout model indicates a likely role in spermatogenesis but not in brain development. Plos One. 12: e0174264
Martinez-Garay I, Guidi LG, Holloway ZG, et al. (2016) Normal radial migration and lamination are maintained in dyslexia-susceptibility candidate gene homolog Kiaa0319 knockout mice. Brain Structure & Function
Martinez-Garay I, Gil-Sanz C, Franco SJ, et al. (2016) Cadherin2/4-signaling via PTP1B and catenins is critical for nucleokinesis during radial neuronal migration in the neocortex. Development (Cambridge, England)
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