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Tyler Cutforth

Affiliations: 
Stanford University, Palo Alto, CA 
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"Tyler Cutforth"
Mean distance: 13.44 (cluster 11)
 
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Cross-listing: FlyTree

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Publications

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Platt MP, Bolding KA, Wayne CR, et al. (2020) Th17 lymphocytes drive vascular and neuronal deficits in a mouse model of postinfectious autoimmune encephalitis. Proceedings of the National Academy of Sciences of the United States of America
Mazzoni J, Smith JR, Shahriar S, et al. (2017) The Wnt Inhibitor Apcdd1 Coordinates Vascular Remodeling and Barrier Maturation of Retinal Blood Vessels. Neuron
Platt MP, Agalliu D, Cutforth T. (2017) Hello from the Other Side: How Autoantibodies Circumvent the Blood-Brain Barrier in Autoimmune Encephalitis. Frontiers in Immunology. 8: 442
Roh JD, Choi SY, Cho YS, et al. (2017) Increased Excitatory Synaptic Transmission of Dentate Granule Neurons in Mice Lacking PSD-95-Interacting Adhesion Molecule Neph2/Kirrel3 during the Early Postnatal Period. Frontiers in Molecular Neuroscience. 10: 81
Cutforth T, DeMille MM, Agalliu I, et al. (2016) CNS autoimmune disease after Streptococcus pyogenes infections: animal models, cellular mechanisms and genetic factors. Future Neurology. 11: 63-76
Martin EA, Muralidhar S, Wang Z, et al. (2015) The intellectual disability gene Kirrel3 regulates target-specific mossy fiber synapse development in the hippocampus. Elife. 4
Choi SY, Han K, Cutforth T, et al. (2015) Mice lacking the synaptic adhesion molecule Neph2/Kirrel3 display moderate hyperactivity and defective novel object preference. Frontiers in Cellular Neuroscience. 9: 283
Mazzoni J, Cutforth T, Agalliu D. (2015) Dissecting the Role of Smooth Muscle Cells versus Pericytes in Regulating Cerebral Blood Flow Using In Vivo Optical Imaging. Neuron. 87: 4-6
Lengfeld J, Cutforth T, Agalliu D. (2014) The role of angiogenesis in the pathology of multiple sclerosis. Vascular Cell. 6: 23
Prince JE, Brignall AC, Cutforth T, et al. (2013) Kirrel3 is required for the coalescence of vomeronasal sensory neuron axons into glomeruli and for male-male aggression. Development (Cambridge, England). 140: 2398-408
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