Emily L. Newman
Affiliations: | Tufts University, Boston |
Area:
Behavioral NeuroscienceGoogle:
"Emily Newman"Mean distance: (not calculated yet)
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Publications
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| Bordes J, Miranda L, Reinhardt M, et al. (2023) Automatically annotated motion tracking identifies a distinct social behavioral profile following chronic social defeat stress. Nature Communications. 14: 4319 |
| Al-Kachak A, Fulton SL, Farrelly LA, et al. (2023) Histone H3 serotonylation dynamics in dorsal raphe nucleus contribute to stress-induced gene expression and behavior. Biorxiv : the Preprint Server For Biology |
| Narendra S, Klengel C, Hamzeh B, et al. (2022) Genome-wide transcriptomics of the amygdala reveals similar oligodendrocyte-related responses to acute and chronic alcohol drinking in female mice. Translational Psychiatry. 12: 476 |
| Miczek KA, DiLeo A, Newman EL, et al. (2021) Neurobiological Bases of Alcohol Consumption After Social Stress. Current Topics in Behavioral Neurosciences |
| Newman EL, Covington HE, Leonard MZ, et al. (2021) Hypoactive Thalamic Crh+ Cells in a Female Mouse Model of Alcohol Drinking After Social Trauma. Biological Psychiatry |
| Covington HE, Newman EL, Leonard MZ, et al. (2019) Translational models of adaptive and excessive fighting: an emerging role for neural circuits in pathological aggression. F1000research. 8 |
| Newman EL, Covington HE, Suh J, et al. (2019) Fighting Females: Neural and Behavioral Consequences of Social Defeat Stress in Female Mice. Biological Psychiatry |
| Newman EL, Leonard MZ, Arena DT, et al. (2018) Social defeat stress and escalation of cocaine and alcohol consumption: Focus on CRF. Neurobiology of Stress. 9: 151-165 |
| Covington HE, Newman EL, Tran S, et al. (2018) The Urge to Fight: Persistent Escalation by Alcohol and Role of NMDA Receptors in Mice. Frontiers in Behavioral Neuroscience. 12: 206 |
| Newman EL, Albrechet-Souza L, Andrew PM, et al. (2018) Persistent escalation of alcohol consumption by mice exposed to brief episodes of social defeat stress: suppression by CRF-R1 antagonism. Psychopharmacology |