Bruno Gavillet

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2002-2007 Pharmacology and Toxicology University of Lausanne 
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Mukherjee R, Mareninova OA, Odinokova IV, et al. (2015) Mechanism of mitochondrial permeability transition pore induction and damage in the pancreas: inhibition prevents acute pancreatitis by protecting production of ATP. Gut
Rougier JS, Gavillet B, Abriel H. (2013) Proteasome inhibitor (MG132) rescues Nav1.5 protein content and the cardiac sodium current in dystrophin-deficient mdx (5cv) mice. Frontiers in Physiology. 4: 51
Albesa M, Grilo LS, Gavillet B, et al. (2011) Nedd4-2-dependent ubiquitylation and regulation of the cardiac potassium channel hERG1. Journal of Molecular and Cellular Cardiology. 51: 90-8
Leoni AL, Gavillet B, Rougier JS, et al. (2010) Variable Na(v)1.5 protein expression from the wild-type allele correlates with the penetrance of cardiac conduction disease in the Scn5a(+/-) mouse model. Plos One. 5: e9298
El-Haou S, Balse E, Neyroud N, et al. (2009) Kv4 potassium channels form a tripartite complex with the anchoring protein SAP97 and CaMKII in cardiac myocytes. Circulation Research. 104: 758-69
Jespersen T, Gavillet B, van Bemmelen MX, et al. (2006) Cardiac sodium channel Na(v)1.5 interacts with and is regulated by the protein tyrosine phosphatase PTPH1. Biochemical and Biophysical Research Communications. 348: 1455-62
Gavillet B, Rougier JS, Domenighetti AA, et al. (2006) Cardiac sodium channel Nav1.5 is regulated by a multiprotein complex composed of syntrophins and dystrophin Circulation Research. 99: 407-414
Boixel C, Gavillet B, Rougier JS, et al. (2006) Aldosterone increases voltage-gated sodium current in ventricular myocytes. American Journal of Physiology. Heart and Circulatory Physiology. 290: H2257-66
Rougier JS, van Bemmelen MX, Bruce MC, et al. (2005) Molecular determinants of voltage-gated sodium channel regulation by the Nedd4/Nedd4-like proteins. American Journal of Physiology. Cell Physiology. 288: C692-701
Abriel H, Schläpfer J, Keller DI, et al. (2004) Molecular and clinical determinants of drug-induced long QT syndrome: an iatrogenic channelopathy. Swiss Medical Weekly. 134: 685-94
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