Preston Ge

2014-2017 Institute for Cell Engineering Johns Hopkins University School of Medicine, Baltimore, MD, United States 
 2017-2025 Harvard Medical School, Boston, MA, United States 
Parkinson's disease
"Preston Ge"
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Panicker N, Ge P, Dawson VL, et al. (2021) The cell biology of Parkinson's disease. The Journal of Cell Biology. 220
Ge P, Dawson VL, Dawson TM. (2020) PINK1 and Parkin mitochondrial quality control: a source of regional vulnerability in Parkinson's disease. Molecular Neurodegeneration. 15: 20
Brahmachari S, Lee S, Kim S, et al. (2019) Parkin interacting substrate zinc finger protein 746 is a pathological mediator in Parkinson's disease. Brain : a Journal of Neurology
Brahmachari S, Karuppagounder SS, Ge P, et al. (2017) c-Abl and Parkinson's Disease: Mechanisms and Therapeutic Potential. Journal of Parkinson's Disease. 7: 589-601
Mao X, Ou MT, Karuppagounder SS, et al. (2016) Pathological α-synuclein transmission initiated by binding lymphocyte-activation gene 3. Science (New York, N.Y.). 353
Brahmachari S, Ge P, Lee SH, et al. (2016) Activation of tyrosine kinase c-Abl contributes to α-synuclein-induced neurodegeneration. The Journal of Clinical Investigation
Karuppagounder SS, Xiong Y, Lee Y, et al. (2016) LRRK2 G2019S Transgenic Mice Display Increased Susceptibility to 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-Mediated Neurotoxicity. Journal of Chemical Neuroanatomy
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