Juha Partanen

Affiliations: 
University of Helsinki, Helsingfors, Finland 
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"Juha Partanen"
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DeSalvo J, Ban Y, Li L, et al. (2021) ETV4/5 drive synovial sarcoma through control of the cell cycle and the DUX4 embryonic pathway. The Journal of Clinical Investigation
Morello F, Voikar V, Parkkinen P, et al. (2020) ADHD-like behaviors caused by inactivation of a transcription factor controlling the balance of inhibitory and excitatory neuron development in the mouse anterior brainstem. Translational Psychiatry. 10: 357
Morello F, Borshagovski D, Survila M, et al. (2020) Molecular Fingerprint and Developmental Regulation of the Tegmental GABAergic and Glutamatergic Neurons Derived from the Anterior Hindbrain. Cell Reports. 33: 108268
Mennesson M, Orav E, Gigliotta A, et al. (2020) Kainate Receptor Auxiliary Subunit NETO2-Related Cued Fear Conditioning Impairments Associate with Defects in Amygdala Development and Excitability. Eneuro. 7
Ryazantseva M, Englund J, Shintyapina A, et al. (2020) Kainate receptors regulate development of glutamatergic synaptic circuitry in the rodent amygdala. Elife. 9
Mennesson M, Rydgren E, Lipina T, et al. (2019) Kainate receptor auxiliary subunit NETO2 is required for normal fear expression and extinction. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology
Brodski C, Blaess S, Partanen J, et al. (2019) Crosstalk of Intercellular Signaling Pathways in the Generation of Midbrain Dopaminergic Neurons In Vivo and from Stem Cells. Journal of Developmental Biology. 7
Balboa D, Saarimäki-Vire J, Borshagovski D, et al. (2018) Insulin mutations impair beta-cell development in a patient-derived iPSC model of neonatal diabetes. Elife. 7
Zega K, Jovanovic VM, Vitic Z, et al. (2018) Corrigendum: Dusp16 Deficiency Causes Congenital Obstructive Hydrocephalus and Brain Overgrowth by Expansion of the Neural Progenitor Pool. Frontiers in Molecular Neuroscience. 11: 29
Balboa D, Saarimäki-Vire J, Borshagovski D, et al. (2018) Author response: Insulin mutations impair beta-cell development in a patient-derived iPSC model of neonatal diabetes Elife
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