Bojana Zupan

Neuroscience and Behavior Vassar College, Poughkeepsie, NY, United States 
"Bojana Zupan"
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Andre Walcott research assistant 2011-2012 Bowdoin College
Duong T. Chu research assistant 2013-2015 Vassar
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Abbott GW, Tai KK, Neverisky DL, et al. (2014) KCNQ1, KCNE2, and Na+-coupled solute transporters form reciprocally regulating complexes that affect neuronal excitability. Science Signaling. 7: ra22
Liu B, Zupan B, Laird E, et al. (2014) Maternal hematopoietic TNF, via milk chemokines, programs hippocampal development and memory. Nature Neuroscience. 17: 97-105
Zupan B, Toth M. (2012) Fmr-1 as an offspring genetic and a maternal environmental factor in neurodevelopmental disease. Results and Problems in Cell Differentiation. 54: 243-53
Rajadhyaksha AM, Ra S, Kishinevsky S, et al. (2012) Behavioral characterization of cereblon forebrain-specific conditional null mice: a model for human non-syndromic intellectual disability. Behavioural Brain Research. 226: 428-34
Gleason G, Zupan B, Toth M. (2011) Maternal genetic mutations as gestational and early life influences in producing psychiatric disease-like phenotypes in mice. Frontiers in Psychiatry. 2: 25
Gleason G, Liu B, Bruening S, et al. (2010) The serotonin1A receptor gene as a genetic and prenatal maternal environmental factor in anxiety. Proceedings of the National Academy of Sciences of the United States of America. 107: 7592-7
Oh JE, Zupan B, Gross S, et al. (2009) Paradoxical anxiogenic response of juvenile mice to fluoxetine. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology. 34: 2197-207
Baudy RB, Butera JA, Abou-Gharbia MA, et al. (2009) Prodrugs of perzinfotel with improved oral bioavailability. Journal of Medicinal Chemistry. 52: 771-8
Sun Y, Zupan B, Raaka BM, et al. (2009) TRH-receptor-type-2-deficient mice are euthyroid and exhibit increased depression and reduced anxiety phenotypes. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology. 34: 1601-8
Zupan B, Toth M. (2008) Inactivation of the maternal fragile X gene results in sensitization of GABAB receptor function in the offspring. The Journal of Pharmacology and Experimental Therapeutics. 327: 820-6
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