Sylvie Liabeuf
Affiliations: | Aix Marseille 2 |
Area:
Kcc2, Nkcc1, Plasticity, Development, Spinal cord injury, Intracellular trafficking, Bdnf, Serotonin, PhosphorylationGoogle:
"Sylvie Liabeuf"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Laurent Vinay | grad student | 2011 | Aix Marseille 2 | |
| (Plasticités développementale et post-lésionnelle des co-transporteurs cation-chlorure KCC2 et NKCC1 dans la moelle épinière) | ||||
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Publications
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| Pajer K, Bellák T, Grósz T, et al. (2023) Riluzole treatment modulates KCC2 and EAAT-2 receptor expression and Ca accumulation following ventral root avulsion injury. European Journal of Cell Biology. 102: 151317 |
| Caravagna C, Casciato A, Coq JO, et al. (2022) Prenatal Hypoxia Induces Cl Cotransporters KCC2 and NKCC1 Developmental Abnormality and Disturbs the Influence of GABA and Glycine Receptors on Fictive Breathing in a Newborn Rat. Frontiers in Physiology. 13: 786714 |
| Filipchuk A, Pambo-Pambo A, Gaudel F, et al. (2021) Early hypoexcitability in a subgroup of spinal motoneurons in superoxide dismutase 1 transgenic mice, a model of amyotrophic lateral sclerosis. Neuroscience |
| Bras H, Liabeuf S. (2020) Differential effects of spinal cord transection on glycinergic and GABAergic synaptic signaling in sub-lesional lumbar motoneurons. Journal of Chemical Neuroanatomy. 101847 |
| Plantier V, Sanchez-Brualla I, Dingu N, et al. (2019) Calpain fosters the hyperexcitability of motoneurons after spinal cord injury and leads to spasticity. Elife. 8 |
| Plantier V, Sanchez-Brualla I, Dingu N, et al. (2019) Author response: Calpain fosters the hyperexcitability of motoneurons after spinal cord injury and leads to spasticity Elife |
| Coq JO, Delcour M, Ogawa Y, et al. (2018) Mild Intrauterine Hypoperfusion Leads to Lumbar and Cortical Hyperexcitability, Spasticity, and Muscle Dysfunctions in Rats: Implications for Prematurity. Frontiers in Neurology. 9: 423 |
| Sánchez-Brualla I, Boulenguez P, Brocard C, et al. (2017) Activation of 5-HT2A receptors restores KCC2 function and reduces neuropathic pain after spinal cord injury. Neuroscience |
| Liabeuf S, Stuhl-Gourmand L, Gackière F, et al. (2017) PROCHLORPERAZINE INCREASES KCC2 FUNCTION AND REDUCES SPASTICITY AFTER SPINAL CORD INJURY. Journal of Neurotrauma |
| Brocard C, Plantier V, Boulenguez P, et al. (2016) Cleavage of Na(+) channels by calpain increases persistent Na(+) current and promotes spasticity after spinal cord injury. Nature Medicine. 22: 404-11 |